Search or ask a question

How many neurons are in the brain of a monkey?

Receptive field

Superior colliculus

Temporal cortex

Answers from top 6 papers

PDF

Open Access

More filters

Papers (6)	Insight
Journal Article•DOI Localization of GABA-like immunoreactivity in the monkey amygdala. Alexander J. McDonald, J.R. Augustine - Show less +1 more 01 Jan 1993-Neuroscience 212 Citations	This study indicates that neuronal subpopulations in each of the amygdaloid nuclei of the monkey are GABAergic.
Book Chapter•DOI Sensory organization of the superior colliculus in cat and monkey. Mark T. Wallace, Barry E. Stein - Show less +1 more 01 Jan 1996-Progress in Brain Research 72 Citations	These findings suggest that, despite differences in the ontogeny and behavioral repertoires of cat and monkey, there is a substantial conservation in the organization and functional properties of SC neurons.
Journal Article•DOI Electrical activity of the isolated macaque brain. L.C. Massopust, Robert J. White, L.R. Wolin, Maurice S. Albin, Yashon D, N. Taslitz - Show less +5 more 01 Oct 1968-Experimental Neurology 31 Citations	It appears that the isolated primate brain shows a range of electrophysiological activity including states characteristic of the aroused brain of an intact monkey.
Open access•Journal Article•DOI A parieto-frontal network for visual numerical information in the monkey Andreas Nieder, Earl K. Miller - Show less +1 more 11 May 2004-Proceedings of the National Academy of Sciences of the United States of America 482 Citations	This finding suggests a parieto-frontal network for numerosity in monkeys and establishes homologies between the monkey and human brain.
Journal Article•DOI Single-cell recording from the brain of freely moving monkeys. Nandor Ludvig, Juan M. Botero, Hai M Tang, Baiju C. Gohil, John G. Kral - Show less +4 more 30 Apr 2001-Journal of Neuroscience Methods 23 Citations	Using this model, new insights can be obtained into the unique firing repertoire of the neurons of the primate brain.
Journal Article•DOI A comparison of somatostatin and neuropeptide Y distribution in monkey brain M. Flint Beal, Michael F. Mazurek, Joseph B. Martin - Show less +2 more 10 Mar 1987-Brain Research 47 Citations	The distribution of both neuropeptides in monkey brain closely resembles that in human brain.

My columns

Related Questions

How many neurons are in the ape brain?5 answers

How many neurons are in the brains?6 answers

How many neurons are there in a human brain?4 answers

How many neurons do monkeys have?5 answers

How many neurons does a human brain have?3 answers

How many neurons is the human brain?4 answers

See what other people are reading

Do modern convolutional neural networks have sufficient biological fidelity to be a good model for the brain?

Does childhood trauma impact the prefrontal cortex?

How does rotating enrichment items affect habituation in captive animals?

Rotating enrichment items is a critical strategy in preventing habituation and maintaining the effectiveness of enrichment programs for captive animals. Habituation to enrichment items can lead to a decrease in the items' ability to stimulate and engage animals, potentially negating the benefits of enrichment efforts. The contexts provided offer insights into various aspects of this issue. Research on captive animals, ranging from primates to laboratory mice, has shown that introducing variety through rotation of enrichment items can significantly enhance the welfare and behavioral health of these animals. For instance, studies on chimpanzees have highlighted individual preferences for enrichment objects, suggesting that rotating items could cater to varying needs and prevent boredom. Similarly, in laboratory settings, mice showed preferences for different types of enrichment, indicating that rotation could sustain engagement and welfare benefits. In the case of captive sun bears, environmental enrichments that were rotated effectively reduced stereotypic behaviors and cortisol levels, demonstrating a decrease in stress. This suggests that rotating enrichment items can mitigate the risk of habituation and maintain the enrichment's effectiveness in reducing stress. For avian species, like red-tailed black cockatoos, foraging-based enrichments that were varied helped decrease the expression of oral repetitive behaviors, further supporting the importance of rotation in enrichment programs. Moreover, a study on New World primates showed that different olfactory enrichments elicited varied responses, indicating that rotating these items could maintain interest and activity levels without significant habituation over the study period. This is echoed in the management of Asiatic lions, where rotating feeding, sensory, and manipulable enrichment interventions led to improved welfare indices, including reduced stress levels and aberrant behaviors. Lastly, in the context of intensive pig farming, rotating enrichment items like pine and scented wood in confinement stalls was shown to alleviate chronic stress and stereotypical behaviors in sows. Collectively, these studies underscore the importance of rotating enrichment items to prevent habituation, thereby ensuring the continued effectiveness of enrichment programs in enhancing the welfare of captive animals across a variety of species.How does the zona incerta control anxiety?

The zona incerta (ZI) plays a crucial role in modulating anxiety through various mechanisms. Studies have shown that silencing ZI blocks the expression of stress-induced social conditioned place aversion (CPA) and enhances anxiety levels in mice subjected to repeated social defeat stress (RSDS). Dysregulation of microglial engulfment capacity in the ventral zona incerta (ZIV) leads to anxiety-like behaviors in acute pain conditions, with chemogenetic activation of ZIVGABA relieving these symptoms. Furthermore, ZI activation suppresses fear generalization, while chemogenetic inhibition of ZI results in fear generalization, indicating its role in regulating fear responses. Additionally, different subpopulations of ZI neurons, such as SOM, CR, and Vglut2-expressing cells, encode and modulate distinct components of anxiety-related behaviors, with specific neuronal activations inducing or reducing anxiety levels. Overall, the ZI exerts control over anxiety through intricate neural circuits and interactions with microglial cells.Does the presence of a goalkeeper have a significant impact on the accuracy of penalty kicks in soccer?

The presence of a goalkeeper significantly impacts the accuracy of penalty kicks in soccer. Research has shown that the interaction between kickers and goalkeepers plays a crucial role in predicting the direction of the kick, affecting the zone or sector of the goal aimed at. Studies utilizing functional near-infrared spectroscopy (fNIRS) have revealed that players experience different brain activations based on the presence and type of goalkeeper, with anxiety levels affecting performance. Moreover, experiments focusing on shooting performance have demonstrated that providing specific attentional and motivational instructions, especially in combination with an external focus and autonomy support, can enhance the number of goals scored and kicking accuracy when facing a goalkeeper.How does the type and amount of alcohol consumed impact an individual's ability to respond quickly?

The type and amount of alcohol consumed can significantly impact an individual's ability to respond quickly. Alcohol consumption affects neurotransmitter levels in the brain, particularly increasing GABAergic signaling and decreasing glutamatergic signaling, which can modulate executive functioning and impair response inhibition. Studies have shown that alcohol-induced response inhibition deficits are influenced by the degree of response automatization, with impairments occurring mainly in automatized responses. Additionally, the amount of consumed alcohol has been found to have a direct correlation with the driver's reaction time, significantly affecting road safety by influencing stopping distances. Chronic, excessive alcohol use is linked to significant cognitive impairment, particularly in older individuals, who may be at a greater risk due to the cognitive decline associated with aging.How fast synaptogenesis occur?

Synaptogenesis occurs at varying speeds depending on the brain region and developmental stage. In the macaque monkey neocortex, the most rapid phase of synaptogenesis lasts around 40 days, centered on birth, with phase 3 being particularly modifiable by stimulation or deprivation. In the hippocampus, synaptogenesis in CA1 occurs within 30 minutes after long-term potentiation induction, with subsequent changes in presynaptic vesicles and axonal boutons observed at 2 hours. Human fetal cortical synaptogenesis initiates between the 6th and 7th gestational week, with a rapid increase in synaptic density by 12-14 weeks. The adult dentate gyrus displays asynchrony in synapse formation, with GABAergic inputs developing early but slowly, while glutamatergic inputs have a late onset and rapid maturation. These findings collectively highlight the dynamic and region-specific nature of synaptogenesis timelines.What is the difference between fovea centralis and peripheral retina?

The fovea centralis and peripheral retina exhibit distinct characteristics. The fovea, with its high-acuity central vision, contains cone photoreceptors densely packed in a unique array, leading to slower cone signals compared to the peripheral retina. In contrast, the peripheral retina shows asymmetries in visual discrimination, with better sensitivity along the horizontal meridian and lower visual field. Moreover, the foveal optic radiations demonstrate higher fractional anisotropy and mean kurtosis, indicating denser nerve fiber populations compared to the peripheral optic radiations. Additionally, hue-scaling data reveals that peripheral stimuli appear more saturated than foveal stimuli, especially in the green-yellow region, suggesting shifts in unique hue loci with retinal location. These differences highlight the specialized functions and adaptations of the fovea centralis and peripheral retina in visual processing.What are the functions of microglia specially in T. gondii infection?

Microglia play crucial roles in Toxoplasma gondii infection by regulating immune responses and parasite control in the central nervous system (CNS). They exhibit a transcriptional shift to a disease-associated phenotype, express anti-parasitic molecules, and contribute to eliminating T. gondii in a cell-intrinsic manner. Microglia release the alarmin IL-1α, promoting neuroinflammation and aiding in parasite control during chronic T. gondii infection. Additionally, microglia exhibit enhanced migration and hypermotility in response to T. gondii, potentially facilitating parasite dissemination in the brain parenchyma. Overall, microglia's functions in T. gondii infection involve immune regulation, anti-parasitic activity, cytokine release, and modulation of migration, highlighting their critical role in CNS defense against this intracellular parasite.Do monkeys recognize credit ?

Monkeys demonstrate various cognitive abilities related to recognition. They possess a highly developed capacity for face recognition, similar to humans, as shown in studies with capuchin monkeys. Additionally, research on rhesus macaques indicates that neuronal activity in the dorsolateral prefrontal cortex reflects information necessary for credit assignment, linking causes and effects during learning tasks. While monkeys may not recognize credit in the same complex way as humans, they exhibit the ability to recognize token validity and engage in exchanges, resembling some features of human monetary systems. Therefore, monkeys showcase recognition abilities in different contexts, from identifying familiar individuals in pictures to understanding the outcomes of their choices in tasks, indicating a level of cognitive recognition akin to credit assignment.What is the impact of exercise intensity on cortical activity?

Different exercise intensities have varying impacts on cortical activity. Moderate to high-intensity exercises have been shown to increase prefrontal cortex (PFC) activation during cognitive tasks, indicating improved neural processing. Specifically, high-intensity aerobic exercise has been linked to increased cortical activation in the frontal lobe, without affecting temporal lobe activity. Additionally, high-intensity interval training has demonstrated increased corticospinal excitability and reduced inhibition, potentially enhancing motor learning through cortical neuroplasticity. Furthermore, high-intensity exercise training programs have shown to enhance PFC functioning, cognitive performance, cardiac autonomic control, and psychosocial aspects in individuals with a history of drug abuse. Overall, exercise intensity plays a crucial role in modulating cortical activity, with higher intensities often leading to more pronounced effects on neural processing.