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How much time does it take for fat cells to shrink? 

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These results suggest that there is a population of fat cell-precursor cells in the mesenchymal tissue of the new born mouse, and that it takes 2–6 days for the precursor cells to become mature fat cells in the epidiymal fatty tissue.
Compared with centrifuged fat, the squeezed fat and residual fat products exhibited increased specific gravity and increased numbers of adipose-derived stem/stromal cells and endothelial cells per volume, suggesting successful cell/tissue condensation in both squeezed fat and residual tissue of emulsified fat.
These results suggest that mice have mechanisms to maintain total body fat mass that respond to an increase in the number of fat cells present.
These experiments indicated that at least during the early phase of rapid weight gain, accumulation of fat was due to deposition of triglyceride in existing cells rather than to accelerated formation of new fat cells.
When fat is transplanted to a third, common site (beneath the kidney capsule), this difference in size largely disappears, suggesting it is the location of the fat cells rather than of intrinsic differences between the cells that account for their different sizes.
It is supposed that not all unilocular fat cells are terminally differentiated, nor do they lose their proliferative activity.
The fat total cells (CTF) were 1.62 x10(6) cells/mL and presented 98% of viability.
The findings clearly show that the size of the fat cells has a substantial influence on the lipolysis of human fat cells.
The present findings suggest a continuous replacement of brown fat cells by white fat cells during advancing age.
The fat tissue graft might be useful for investigation of the influence of various hormones on human fat cells.
This model can be useful for the study of the metabolic and morphological features of human brown fat cells.
This finding strongly implies an intrinsic adhesiveness between fat-body cells.
These studies highlight the complex cellular components of fat grafts and how they respond differentially to time and collagenase digestion.
This association may be related to the disparate morphology and metabolic behavior of fat cells associated with different body fat distributions.

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