scispace - formally typeset
Search or ask a question

How to connect to WAMP server from another computer? 

Answers from top 8 papers

More filters
Papers (8)Insight
This system has several advantages compared with traditional remote laboratory based on computer server, which are small size, low power consumption, low cost and easy installation and maintenance.
Although network congestion can increase the latencies, WAMP has potential to be used in developing scalable real-time remote ECG monitoring platforms.
The results obtained contribute to our knowledge of the biodiversity of wamp genes in Poaceae.
According to the results, WAMP protocol can support real-time ECG streaming with a 250 Hz sampling rate, real-time HRV analyses, and real-time alarm notifications.
The results have shown that WL, WAMP, ZC, and SSC outperform other features.
We determined that salicylic acid, rather than methyl jasmonate, induces expression of genes of the wamp family.
In turn, the resistance to abiotic stress induced by cadmium ions does not appear to be associated with expression of genes of the wamp family.
The results support the protective role of wamp genes in the response of wheat plants to infections by pathogens.

See what other people are reading

Is there a relationship between meat quality and miR-181a expression level in sheep?
5 answers
What is the relative Protein expression of CES-1in organs?
5 answers
The relative protein expression of CES-1 varies across different organs. In human liver samples, isoforms 1 and 2 are the major forms of CES1 protein, contributing 73-90% of the total expression, with isoforms 3 and 4 showing impaired activity. CESP-1, a corneal endothelium-specific protein, is expressed in the corneal epithelium, corneal endothelium, brain, testis, and ovary, with subcellular localization in the mitochondria of cultured human corneal endothelium. CES1 and CES2 expression and activity are age-dependent, with lower levels in newborns that increase with age, showing significant differences in expression between age groups. A study utilizing allele-specific protein expression assays identified cis-acting regulatory variants significantly associated with CES1 protein expression and catalytic activity in human livers.
Is respiratory secretory cell associated with lung cancer?
4 answers
Respiratory secretory cells play a significant role in lung cancer. Secretory leukocyte protease inhibitor (SLPI), a serine protease inhibitor primarily produced in the respiratory tract, is elevated in lung cancer. Additionally, lung cancer cells secrete proteins that can be nephrotoxic, potentially leading to kidney failure as a rare complication in lung cancer patients. Furthermore, secretory proteins have been identified as a rich source for biomarker research in lung adenocarcinoma (LUAD), aiding in the early diagnosis of this type of lung cancer. Moreover, a case study highlighted the presence of secretory component (SC) in lung adenocarcinoma cells, suggesting that these cancer cells produce and secrete SC, contributing to high serum levels of secretory IgA (sIgA). Therefore, respiratory secretory cells are indeed associated with lung cancer, influencing its progression and diagnostic biomarkers.
Do enhancers recognize chomatin modifications or promotor sequences`?
5 answers
Enhancers primarily recognize chromatin modifications rather than promoter sequences. Studies have shown that histone modifications at enhancers are predictive of gene expression, similar to promoters, indicating their role in transcriptional regulation. Additionally, enhancer-promoter interactions (EPIs) are influenced by DNA binding motifs of transcription factors like CTCF, which affect the expression of target genes. Computational methods like SEPT and EPIVAN leverage DNA sequences to predict enhancer-promoter interactions, highlighting the importance of sequence-level information in identifying EPIs. These findings collectively suggest that enhancers primarily recognize chromatin modifications and DNA binding motifs of transcription factors rather than promoter sequences in regulating gene expression.
What physical and chemical processes lead to the development of tangled roots in citrus plants?
5 answers
The development of tangled roots in citrus plants is influenced by various physical and chemical processes. Root anatomy plays a crucial role in this phenomenon, as observed in studies focusing on the differentiation of root layers. Additionally, the anisotropy of root growth, driven by complex interactions involving phytohormones like auxin, cell growth, and cytoskeleton rearrangements, contributes to the formation of tangled roots. Furthermore, the expression of cytokinin response regulators (CcRRs) in different root zones affects root growth and development, potentially leading to tangled root structures. These studies highlight the intricate mechanisms involving root anatomy, hormonal signaling, and gene expression that collectively contribute to the development of tangled roots in citrus plants.
AMR genes Biofilm formation Cathelicidin S aureus
5 answers
Staphylococcus aureus utilizes a set of resistant genes to combat the threat posed by antimicrobial peptides like cathelicidin LL-37. Biofilm formation is a crucial aspect of S. aureus survival and antimicrobial resistance, with biofilm-related proteins playing a significant role. Studies on multidrug-resistant S. aureus strains revealed that biofilm formation is prevalent among these strains, impacting their ability to persist and infect. Additionally, S. aureus isolates exhibit resistance to various antibiotics, with a high percentage showing biofilm production, indicating their persistence in clinical settings. The interplay between AMR genes, biofilm formation, and cathelicidin resistance underscores the complex mechanisms employed by S. aureus to evade antimicrobial threats and highlights the importance of understanding these interactions for developing effective treatment strategies.
AMR genes Biofilm formation Cathelicidin S aureus in Silico
5 answers
In silico analysis of AMR genes, biofilm formation, and the interaction with cathelicidin in Staphylococcus aureus reveals crucial insights. Studies highlight the complexity of S. aureus resistance mechanisms, including membrane alterations to combat antimicrobial peptides. Biofilm formation is a key survival strategy for S. aureus, aiding in environmental persistence and antimicrobial resistance. The presence of biofilm-related proteins and specific genes like icaD play significant roles in biofilm development and antibiotic resistance. Additionally, the interaction between S. aureus and cathelicidin peptides underscores the importance of understanding peptide-response genes for infection, biofilm formation, and antibiotic design. Integrating these findings through in silico analysis can provide a comprehensive understanding of AMR mechanisms, biofilm dynamics, and peptide interactions in S. aureus.
How do secondary metabolites and peptides disrupt the integrity of Salmonella biofilms?
4 answers
Secondary metabolites and peptides play a crucial role in disrupting the integrity of Salmonella biofilms. These compounds have been shown to impact biofilm formation and maintenance through various mechanisms. For instance, secondary metabolites produced during biofilm growth can influence the regulation of genes involved in biofilm formation and competition. Additionally, antibiofilm agents, including peptides and secondary metabolites, have been investigated for their efficacy in inhibiting biofilm formation by clinically relevant pathogens. Moreover, extracellular DNA (eDNA) released from biofilm cells, along with genomic DNA (gDNA), can significantly impact biofilm formation, with enzymatic digestion of DNA resulting in decreased biofilm formation. Overall, secondary metabolites and peptides derived from natural products have shown promise in combating microbial biofilms, including those formed by Salmonella.
What are the technical limitations of using reverse transcription polymerase chain reaction (RT-PCR) for gene expression analysis?
4 answers
The technical limitations of using reverse transcription polymerase chain reaction (RT-PCR) for gene expression analysis include issues such as errors in database entries leading to false sequence information, mismatches between gene, mRNA, and protein sequences, lack of experimental validation in splicing patterns, and the necessity for proper quality control steps throughout the assay to ensure reproducibility and reliability of data. Additionally, the handling of undetermined quantification cycle (Cq) values in RT-qPCR data analysis pipelines can introduce bias and lower precision, impacting downstream differential expression analysis. To overcome these limitations, strategies such as employing multiple loci for bloodmeal analysis and building databases with multiple mitochondrial reference genes for reliable host species identification are recommended.
What is the molecular mechanism of flexible docking followed by rigid docking in plant metabolites antibacterial analysis?
4 answers
Flexible docking followed by rigid docking is a crucial approach in analyzing plant metabolites for antibacterial activity. This methodology involves initially conducting flexible docking to identify potential compounds that interact favorably with specific bacterial protein targets. Subsequently, rigid docking is employed to further refine the interactions and assess the binding affinity of the selected compounds with the target proteins. For instance, in the study of Pyracantha crenulata phytoconstituents, molecular docking revealed that compounds like rutin and phloridzin exhibited superior docking scores against various antibacterial targets. Similarly, in the analysis of Brucea antidysentrica compounds, flexible and rigid docking highlighted the interactions of canthin-6-one and 1,11-dimethoxycanthin-6-one with DNA gyrase enzyme, indicating their potential as antibacterial inhibitors.
Cancer biology regulation variation
4 answers
Variations in gene regulation play a crucial role in cancer biology. While mutations in coding regions were traditionally considered responsible for disease phenotypes, recent research highlights the significance of functional noncoding regions in cancer etiology. Copy number alterations, epigenetic changes, transcription factors, and microRNAs collectively explain a significant portion of gene expression variation in cancer-associated genes. Regulatory networks in cancer genomes exhibit complex combinatorial patterns, modulating gene expression at a finer scale. Noncoding RNAs, especially pseudogenes, are found to strongly activate the cancer genome and induce local targets, shifting the regulatory regime towards a cancerous niche. Understanding these regulatory alterations is crucial for unraveling the initiation and progression of cancer, offering insights for future diagnostic and prognostic tools.