Search or ask a question

How to make antioxidant tea?

Oxidative stress

Lipid peroxidation

Hydroxybenzoic acid

Camellia sinensis

Answers from top 5 papers

PDF

Open Access

More filters

Papers (5)	Insight
Journal Article•DOI The effect of tea consumption on oxidative stress in smokers and nonsmokers. James E. Klaunig, Yong Xu, Chi Han, Lisa M. Kamendulis, Junshi Chen, Carla Heiser, Michael S. Gordon, Emile R. Mohler - Show less +7 more 01 Apr 1999-Experimental Biology and Medicine 103 Citations	The beneficial properties of tea to human health may be related to the antioxidant properties of tea components.
Journal Article•DOI Antioxidant activity of various extracts of old tea leaves and black tea wastes (Camellia sinensis L.) Reza Farhoosh, Gholam A. Golmovahhed, Mohammad Hossein Haddad Khodaparast - Show less +2 more 01 Jan 2007-Food Chemistry 137 Citations	This suggested that the antioxidant activity of tea extracts likely involves other mechanisms in addition to those of reductones.
Open access•Journal Article•DOI Determination of Antioxidant Activity in Tea Extracts, and Their Total Antioxidant Content Alexander Yashin, Yakov I. Yashin, Boris Nemzer - Show less +2 more 01 Oct 2011-American Journal of Biomedical Sciences 68 Citations	Data gathered about antioxidant content of these different tea samples can be used to estimate quality and type of tea.
Book Chapter•DOI Tea Processing and its Impact on Catechins, Theaflavin and Thearubigin Formation Anakalo Shitandi, Francis Muigai Ngure, Symon M. Mahungu - Show less +2 more 01 Jan 2013-Tea in Health and Disease Prevention 13 Citations	Optimal clone-specific processing conditions can therefore be utilized to add value to black tea with regard to antioxidant activity.
Journal Article•DOI Temperature and Time of Steeping Affect the Antioxidant Properties of White, Green, and Black Tea Infusions Fatemeh Hajiaghaalipour, Junedah Sanusi, M.S. Kanthimathi - Show less +2 more 01 Jan 2016-Journal of Food Science 62 Citations	The results of this study showed the antioxidant capacity of white and green tea was greater than black tea.

My columns

Related Questions

How to make Jun tea?4 answersJun tea can be made using a method that involves several steps. First, the citrus junos peel slices are dried, and then licorice water is injected into the dried peel slices. Next, a decocted ssanghwa herb solution is injected into the peel slices, followed by a second drying step. Finally, the dried peel slices are packed in a tea-bag packing paper. This method allows users to enjoy the functionality of both citrus junos and ssanghwa tea, and it helps to redeem the unique bitter taste of citrus junos through licorice. The resulting tea provides various effects and can be consumed without repulsion, making it suitable for the prevention of flu.

How do you make antioxidant water?4 answers

How to Make an antioxidant serum?5 answers

Which green tea has highest antioxidants?6 answers

Which green tea has more antioxidants?7 answers

Which tea is best for antioxidants?7 answers

See what other people are reading

What is the impact of spraying nano_ nitrogen and potassium on the photosynthesis process in Calendula officinalis plants?

Spraying nano-nitrogen and potassium on Calendula officinalis plants can have varying impacts on the photosynthesis process. Research indicates that the application of cerium oxide nanoparticles (CeO2 NPs) induced changes in physiological parameters, such as chlorophyll contents, in marigold plants. Additionally, the study on the effects of growth stimulants like yeast extract, humic acid, and algae extract showed improvements in physiological characteristics like chlorophyll a, chlorophyll b, and total chlorophyll in leaves when applied to Calendula officinalis plants. Moreover, the application of mineral and organic nitrogen fertilizers, including nano-chelate nitrogen, influenced the antioxidant activity in marigold plants, which is crucial for photosynthesis processes. Therefore, the combined data suggests that spraying nano-nitrogen and potassium can potentially impact the photosynthesis process in Calendula officinalis plants by influencing physiological parameters and antioxidant activity.Chitosan from milkfish scales as bioactive coating against plant disease?

Chitosan derived from milkfish scales can indeed serve as a bioactive coating against plant diseases. Chitosan, a biodegradable and biocompatible compound, exhibits antimicrobial properties that can effectively combat pathogens in plants. Studies have shown that chitosan, particularly when combined with plant extracts, enhances its antimicrobial and antioxidant capabilities, making it a valuable resource for food preservation and disease management in agriculture. Furthermore, chitosan extracted from shellfish has demonstrated significant antifungal potential against various plant pathogens, leading to a reduction in disease severity and incidence in crops like rice. Therefore, utilizing chitosan from milkfish scales as a bioactive coating can be a sustainable and effective strategy to protect plants from diseases.What of chronic kidney disease?

Chronic kidney disease (CKD) is a global health burden affecting neonates to geriatric patients. Neonatal CKD is linked to prematurity, intrauterine growth retardation, and acute kidney injury, leading to long-term complications like anemia, hypertension, and growth restriction. In adults, CKD often stems from diabetes and hypertension, with common features including fibrosis, tubular atrophy, and oxidative stress. CKD progression involves nephron loss, glomerulosclerosis, and podocytopathy, leading to end-stage renal failure. Optimal CKD management necessitates a multidisciplinary approach, emphasizing nutritional therapy and palliative care to maintain quality of life. Understanding the molecular pathways underlying CKD progression is crucial for developing effective therapeutic interventions.How does ACTH cause hyperpigmentation?

ACTH causes hyperpigmentation through its interaction with α-melanocyte-stimulating hormone (α-MSH) and the melanocortin-1 receptor (MC1R). This interaction leads to skin pigmentation by activating the oxidative stress/NF-κB/ACTH/cAMP/tyrosinase pathway. In conditions like ectopic ACTH syndrome, hyperpigmentation is a common symptom, often associated with malignancies. Additionally, in cases of primary adrenal insufficiency post-adrenalectomy, ACTH plays a role in skin hyperpigmentation, which can be reversed by low-dose dexamethasone therapy. Furthermore, rare instances of ectopic ACTH syndrome, originating from tumors like thymic neuroendocrine carcinoma, can also lead to general hyperpigmentation. In summary, ACTH-induced hyperpigmentation is a complex process involving various pathways and interactions, often seen in different pathological conditions.How does Liraglutide affect bilirubin metabolism in the liver?

Liraglutide has a significant impact on bilirubin metabolism in the liver. Studies show that Liraglutide treatment reduces liver damage and fibrosis. It decreases oxidative stress, iron accumulation, and ferroptosis, which are crucial factors affecting liver function. Additionally, Liraglutide improves hepatic steatosis, fibrosis, and autophagy in nonalcoholic fatty liver disease (NAFLD). Moreover, Liraglutide treatment corrects diet-induced alterations in glucose metabolism, reduces hepatic steatosis, and inflammation in nonalcoholic steatohepatitis (NASH). Furthermore, Liraglutide treatment in overweight patients with type 2 diabetes mellitus (T2DM) and NAFLD leads to a reduction in liver fat content and serum fibroblast growth factor 21 (FGF21) levels. Overall, Liraglutide plays a crucial role in improving liver health by modulating various pathways related to liver function and metabolism.What is the knowledge gap in obesity induced kidney injury?

The knowledge gap in obesity-induced kidney injury lies in understanding the specific mechanisms by which obesity contributes to renal damage. While obesity is recognized as a significant risk factor for kidney disease, the exact pathways through which it leads to renal injury remain unclear. Studies have highlighted various factors such as tubuloglomerular feedback response impairment, inflammation, oxidative stress, and lipid metabolism disorders, ATP-citrate lyase induction promoting ectopic lipid accumulation and fibrogenesis, and aberrant hypoxic responses due to dysfunction of PHD2. However, there is a need for further research to elucidate the intricate interplay of these factors and their impact on kidney health in the context of obesity, paving the way for targeted treatments and preventive strategies.What are the benefits of fish oil supplementation?

Fish oil supplementation offers various benefits, as evidenced by research. It has been shown to alleviate muscle damage, inflammation, and redox imbalance induced by intense strength exercises, making it ideal for individuals engaging in strength-training programs. Additionally, fish oil, rich in omega-3 fatty acids, may contribute to reducing the risk of cardiovascular diseases by improving cholesterol efflux capacity, potentially aiding in the prevention of atherosclerosis. Moreover, fish oil has been linked to inhibiting inflammation and oxidative stress pathways, which are crucial in the development and progression of diabetic nephropathy, showcasing its potential therapeutic role in this condition. Furthermore, preliminary data suggests that fish oil supplementation does not impact arterial stiffness or cognitive function in healthy adults, indicating the need for further investigation in this area. Fish oil supplementation may also mitigate inflammatory responses induced by particulate matter exposure through modulation of fatty acid metabolism, highlighting its potential protective effects against environmental pollutants.What is the formation and réactivité of Free radical?

Free radicals are formed through various processes such as the reaction of neurotransmitters with tert-butylperoxy radicals, the interaction of bissiloxydioxasilirane groups with molecules like H2, CH4, and C2H6, and the addition to alkenic double bonds in free-radical polymerization. These radicals exhibit reactivity by covalently modifying lipids, proteins, and DNA in physiological processes like cerebral radical formation. Low-temperature treatment of polymers and monomers with halogens also leads to the spontaneous formation of free radicals, which can initiate polymerization reactions. The reactivity of these radicals is crucial in various biological and chemical processes, influencing disease progression in neurodegenerative conditions like Alzheimer's and Parkinson's disease. Understanding the formation and reactivity of free radicals is essential for elucidating their roles in different systems and diseases.How does the mutation in the hemoglobin gene affect red blood cell structure and function in sickle cell anemia?

The mutation in the hemoglobin gene in sickle cell anemia, characterized by the substitution of valine for glutamine at position 6 of the β-globin chain, leads to the production of sickle hemoglobin (HbS). This mutation causes HbS to polymerize under low-oxygen conditions, resulting in distorted and less elastic red blood cells (RBCs) that are prone to sickling and vaso-occlusion. The sickle RBCs are the primary source of oxidative stress in SCD due to an imbalance between prooxidants and antioxidants, leading to continuous production of reactive oxygen species (ROS) and subsequent membrane alterations, reduced deformability, and release of micro-vesicles. These structural changes in RBCs contribute to the pathophysiology of SCD, causing complications such as hemolysis, vaso-occlusive crises, chronic inflammation, and tissue damage.What is the mTORC1-Plin3 and liver fibrosis-induced muscle atrophy?

mTORC1 signaling activation in aging contributes to muscle atrophy by inducing oxidative stress and fiber damage, with GDF signaling playing a role in this process. On the other hand, liver fibrosis-induced muscle atrophy involves TNFα signaling mediated by circulating factors produced in the damaged liver, leading to skeletal muscle atrophy. Additionally, PNPLA3 polymorphisms influence the relationship between skeletal muscle loss and non-alcoholic fatty liver disease (NAFLD), with low appendicular skeletal muscle mass associated with NAFLD and liver fibrosis, particularly in individuals with specific PNPLA3 genotypes. These findings collectively highlight the intricate interplay between mTORC1 signaling, liver fibrosis, and skeletal muscle atrophy, underscoring the multifactorial nature of muscle wasting in various pathological conditions.What uremic toxin can bind ary hydrocarbon receptor?

Indoxyl sulfate (IS) and indole-3-acetic acid (IAA) are uremic toxins that can bind to the aryl hydrocarbon receptor (AhR). These toxins are known to activate AhR, a ligand-activated transcription factor, which plays a role in various physiological processes. Studies have shown that IS and IAA, derived from gut microbiota metabolism, accumulate in patients with chronic kidney disease (CKD) and can act as potent ligands for AhR. Activation of AhR by these uremic toxins has been linked to oxidative stress, endothelial dysfunction, inflammation, and cardiovascular diseases in CKD patients. Understanding the interaction between uremic toxins like IS and IAA with AhR provides insights into the pathophysiology of CKD-related complications and suggests potential therapeutic targets for managing cardiovascular issues in CKD patients.