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What happens to neurons when you drink too much water? 

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In-Young Choi, Rolf Gruetter 
01 Jan 2012
This is what this neural metabolism in vivo tells you.
As this molecular and cellular physiology of neurons, it will really give you the good idea to be successful.
In general, overcorrection of hyponatremia is caused by either "too much salt (Na(+) + K(+)) gained" or "too much water lost."
First, we show that rats may be induced to drink unpalatable 2.7% NaCl solution in addition to water after repeated amphetamine injection.
This decrease in radon probably happens during the water management and storage of mains drinking water.
Caregiver's responsibility would be to inform athletes about potential dangers of drinking too much water and also advise them to refrain from using hypertonic fluid replacements.
These experiments showed that the apparent sparing of function is incomplete; when tested as adults, the brain-damaged animals did not eat in response to acute glucoprivation, nor did they drink water and saline in response to hypovolemia.
Advice to drink water is associated with reduced weight gain in children and weight loss in dieting adults.
However, they will likely drink less tannin water if normal water is available.
They appear to drink normally to overnight water deprivation but remain in negative water balance because of excessive urinary water loss during the deprivation period.
Thirsty hypothalamic hyperphagic rats are less willing than normals to drink water containing quinine.
This paper summarizes systematic observations confirming that such animals drink more water than controls and additionally suggests that this hyperdipsia is not secondary to involuntary water loss.
Unlike the normal rat which will continue to drink water in the absence of food, the rat with lateral hypothalamic lesions that is no longer aphagic or adipsic will not drink water when it cannot eat, and it will drink less, not more, water in response to direct challenges to the regulation of body water that depress feeding, such as water deprivation, increased serum osmotic pressure, and hyperthermia2.
It was concluded that rats must first drink water in order to reduce their water deficit to some threshold before saccharin is drunk.
The results show that rats drink large volumes of a palatable saccharine solution and rely on the kidneys to eliminate excess water.
In addition, when their urine output is suppressed these diabetic rats drink almost exactly the amount of water needed to dilute a cellular thirst stimulus (hypertonic saline) to isotonicity and drink the same amount as controls after equivalent extracellular thirst stimuli (polyethylene glycol) suggesting that important drinking mechanisms are not located in the median eminence.

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