Showing papers on "Animal mortality published in 1984"
TL;DR: The results suggest that the nutritional status of vitamin E may influence the cellular susceptibility of rats to cigarette smoking.
37 citations
Book•
30 May 1984
TL;DR: In this paper, the authors present a model for dealing with the human/animal bond following the loss of a pet in a veterinary practice, based on the psychosocial aspects of bereavement.
Abstract: Foreword (William J. Kay) Preface (William J. Kay, Austin H. Kutscher) I: The Human/Companion Animal Bond. 1. The Moral Status of Animals (Bernard E. Rollin) 2. Pet Animals and Human Well-being (M.W. Fox) 3. Health Consequences of Pet Ownership (Erika Friedmann, Aaron A. Katcher, Sue A. Thomas, James J. Lynch) 4. Nonconventional Human/Companion Animal Bonds (James M. Harris) 5. When Pet Animals Die (Jacob Antelyes) 6. Population Aspects of Animal Mortality (Alan M. Beck) II: The Grieving Human Companion. 7. Grief at the Loss of a Pet ( Boris M. Levinson) 8. Psychosocial Aspects of bereavement (Herbert A. Nieburg) 9. Relief and Prevention of Grief (Leo K. Bustad, Linda M. Hines) 10. Healing Emotionally Disturbed Children Cope with Loss of a Pet (Mary Link) 11. Development of a Social Work Service to Deal with Grief after Loss of a Pet (Eleanor L. Ryder) 12. Social Work in a Veterinary Hospital: Response to Owner Grief Reactions, (Jamie Quackenbush) 13. Illness and Death of Pets: Role of the Human-Health-Care Team (Michael J. McCulloch) III: Veterinary Medicine Perspectives. 14. The Human/Animal Bond Revisited (Esther Braun) 15. Clinical Aspects of Grief Associated with Loss of a Pet: A Veterinarian's View (Marc A. Rosenberg) 16. Role of the Animal Health Technician in Consoling Bereaved Clients (Sally Oblas Walshaw) 17. Owner/Pet Attachment Despite Behavior Problems (Victoria L. Voith) 18. Owner/Pet Pathologic Attachment: The Veterinarian's Nightmare (E.K. Rynearson) 19. Death of Pets Owned by the Elderly: Implications for Veterinary Practice (George Paulus, John C. Thrush, Cyrus S. Stewart, Patrick Hafner) 20. Psychosocial Model of Veterinary Practice, William H. Sullivan, Carole E. Fudin) 21. Family Psychotherapy Methodology: A Model for Veterinarians and Clinicians (D.T. Wessels Jr.) 22. Epilogue: A Historical Perspective, (Egilde Seravalli) Contributors. Index.
16 citations
TL;DR: It is suggested that depletion of vitamin E renders rats more susceptible to the adverse effect of nitrite, and that nitrite administration potentiates deficiency ofitamin E in rats.
9 citations
TL;DR: It seems that diminished GP activity resulting from Se deficiency and the ensuing lack of increase in NADPH production were poorly correlated with mouse tolerance to O3, which suggests a “mobilization” of Se to the lung under O3 stress, and it is possible that such a mobilization contributes to the lungs reserve of antioxidants, and hence the comparable mortality in both dietary Se regimens.
Abstract: We fed female strain A/St mice selenium (Se) test diets containing either no Se (−Se) or 1 ppm Se (+Se) for 11 wk. Both diets contained 55 ppm vitamin E. We then exposed three groups of mice from each dietary regimen to either 0.8 ppm (1568 μg/m3) O3 (low-level) continuously for 5 d, 10.0 ppm (19,600 μg/m3) O3 (high-level) for 12 h, or filtered room air, where the latter served as a control for both O3 exposures. After O3 exposures we analyzed the lungs for various physical and biochemical parameters, and compared the results to those obtained from the air controls. The results showed that the difference in dietary Se intake produced an eightfold difference in Se content and a three-fold difference in glutathione peroxidase (GP) activity in the lung, but few changes in other lung parameters. With low-level O3 exposure, NADPH production increased significantly in +Se mice, but did not change in −Se mice. With high-level O3 exposure we observed comparable effects for both dietary regimens, including animal mortality, which was 24% for −Se and 14% for +Se mice. Thus, it seems that diminished GP activity resulting from Se deficiency and the ensuing lack of increase in NADPH production were poorly correlated with mouse tolerance to O3. The lung Se content increased in both dietary regimens after O3 exposure, but the increase was greater after high-level O3 exposure. This suggests a “mobilization” of Se to the lung under O3 stress. It is possible that such a mobilization contributes to the lung reserve of antioxidants, and hence the comparable mortality in both dietary Se regimens.
7 citations