Topic
Bufo melanostictus
About: Bufo melanostictus is a research topic. Over the lifetime, 196 publications have been published within this topic receiving 1168 citations.
Papers published on a yearly basis
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TL;DR: Inhibition of spermatogenic activity and increase in Leydig Cell nuclear area, testicular delta 5-3 beta-hydroxysteroid dehydrogenase and 17beta-HSD activities were noted after Bidder's organectomy and it is supposed that estradiol coming from the Bidder’s organ might play a role in normal testicular activities.
Abstract: Inhibition of spermatogenic activity and increase in Leydig Cell nuclear area (L C N A), testicular delta 5-3 beta-hydroxysteroid dehydrogenase (delta 5-3 beta-HSD) and 17 beta-hydroxysteroid dehydrogenase (17 beta-HSD) activities were noted after Bidder's organectomy. Administration of estradiol in Bidder's organectomized toad showed more or less similar result as the control animal. It is supposed that estradiol coming from the Bidder's organ might play a role in normal testicular activities.
3 citations
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TL;DR: The results suggest that vitamin A induces the above mentioned abnormalities by either switching on or over-expressing or disrupting the limb-specific hox genes by yet unknown mechanisms.
Abstract: Several types of limb deformities were induced by vitamin A in B. melanostictus. These ranged from total suppression of all the limbs (ectromelia) to partial development of either the forelimb or the hindlimb or both (mesomelia) to reduction or absence of digits in either the forelimbs or hindlimbs or both and absence of long bones in either the forelimbs or hindlimbs or both (phocomelia) or duplication of the hindlimbs (polymelia). All the limb abnormalities were induced in the developing limbs of the tail amputated tadpoles of B. melanostictus following vitamin A treatment, which is all the more interesting. The results suggest that vitamin A induces the above mentioned abnormalities by either switching on or over-expressing or disrupting the limb-specific hox genes by yet unknown mechanisms.
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TL;DR: It seems that after a short exposure to the toxic toad tadpoles B. melanostictus, predators may learn to decrease going after unpalatable prey, subsequently it seems they may express short-term behavioral memory in order to avoid the toxic prey.
Abstract: We examined the prey selection and behavioral responses of tiger frog
Hoplobatrachus chinensis larvae exposed to unpalatable and palatable
sympatric prey tadpoles, Bufo melanostictus and Pelophylax
nigromaculatus . We found that after a short exposure to the toxic
toad tadpoles B. melanostictus, predators may learn to decrease going
after unpalatable prey, subsequently it seems they may express
short-term behavioral memory in order to avoid the toxic prey. In
general, H. chinensis showed no preference for either any of the two
prey species, which may be the result of P. nigromaculatus using
behavioral performance and chemical defense as antipredatation
strategies. These results facilitate further investigation of other
aspects of the behavioral ecology of these three anuran species and
hint at some potentially interesting possibilities of memory in choice
of prey which may suggest further study.
3 citations