Topic
Coturnix
About: Coturnix is a research topic. Over the lifetime, 953 publications have been published within this topic receiving 23305 citations.
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TL;DR: Genetic variation in Coturnix is discussed with reference to the natural history of the species, a recent demonstration that it is relatively resistant to some pesticides and the problem of “hybrid vigour”, both at the molecular and the population level.
62 citations
TL;DR: The toxicity of dietary lead in Japanese quail was investigated and it was suggested that lead interfered with normal sexual development in the males and prevented normal primary antibody responses to sheep erythrocytes.
61 citations
TL;DR: Comparisons of cell size and number showed that aged sexually active males had fewer, larger AROM-ir cells when compared to young males, suggesting neuroplasticity of specific neural systems and a critical role of estradiol in maintaining reproductive function.
60 citations
TL;DR: It was found that mercury potentiated the toxicity and biochemical effects of parathion and was directly related to the inhibition of brain cholinesterase in birds fed clean feed and those fed morsodren-treated feed.
Abstract: We found that mercury potentiated the toxicity and biochemical effects of parathion. Male Coturnix quail (Coturnix coturnix japonica) were fed a sublethal concentration of morsodren (4 ppm as methyl mercury) for 18 weeks. This resulted in an accumulation of 21.0 ppm of mercury in the liver and 8.4 ppm in the carcass. Birds fed clean feed and those fed morsodren-treated feed were orally dosed with 2, 4, 6, 8, and 10 mg/kg parathion, and their 48-h survival times compared. The computed LD50 was 5.86 mg/kg in birds not fed morsodren and 4.24 in those fed the heavy metal. When challenged with a sublethal, oral dose of parathion (1.0 mg/kg), morsodren-fed birds exhibited significantly greater inhibition of plasma and brain cholinesterase activity than controls dosed with parathion. Brain cholinesterase activity was inhibited 41% in morsodren-fed birds and 26% in clean-fed birds dosed with parathion, which suggested that the increase in parathion toxicity in the presence of morsodren was directly related to the inhibition of brain cholinesterase.
60 citations
TL;DR: These results provide the first unequivocal evidence that short-term prenatal sensory stimulation can affect the development of an avian embryo.
Abstract: A single 2-hour exposure to auditory stimulation at any point during the final 3 days of incubation accelerates the hatching of Japanese quail. The 3-day sensitive period includes both prenatal and perinatal stages of incubation. So far as is known these results provide the first unequivocal evidence that short-term prenatal sensory stimulation can affect the development of an avian embryo.
60 citations