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Cuneate nucleus

About: Cuneate nucleus is a research topic. Over the lifetime, 614 publications have been published within this topic receiving 24859 citations. The topic is also known as: cuneate nucleus of spinal cord.


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Journal ArticleDOI
TL;DR: It is suggested that hypercapnic-hypoxic exposures (modeling prone sleeping or sleep apnea) are more likely to induce NMDA receptor changes in the developing brainstem than nicotine exposure alone.

9 citations

Journal ArticleDOI
TL;DR: Choline chloride administered after HC-3 and high frequency stimulation had resulted in depression, restored transmission through the nucleus and results are consistent with the presence of cholinergic synapses in the cuneate nucleus.
Abstract: SummaryIntravenous injection of HC-3 resulted in depression of potentials in the cuneate nucleus of the cat following high frequency stimulation of the radial nerve. No depression followed low frequency continuous stimulation. Choline chloride administered after HC-3 and high frequency stimulation had resulted in depression, restored transmission through the nucleus. The results are consistent with the presence of cholinergic synapses in the cuneate nucleus.

9 citations

Journal ArticleDOI
TL;DR: In this paper, spontaneous and paroxysmal cerebral cortical synchronized activity was used as reference to study the cortical impact exerted on subcortical neurons, including neuronal populations that originate descending rubrospinal, tectospinal and reticulospinal motor axons.

9 citations

Journal ArticleDOI
TL;DR: Immunohistochemical staining data for AMPA and GABAA/B receptor subunits of area 3b cortex and cuneate nucleus of adult squirrel monkeys one to five years after median and ulnar nerve transection suggest cortical plasticity continues to change over many months as receptive field reorganization occurs, while brainstem plasticity obtains a level of stable persistence by one month.

9 citations


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Performance
Metrics
No. of papers in the topic in previous years
YearPapers
20234
20222
202115
20204
20195
20186