Dysarthria

About: Dysarthria is a research topic. Over the lifetime, 2402 publications have been published within this topic receiving 56554 citations.

The King George VI’s Speech Errors in David Seidler movie “King’s Speech”

Abstract: According to the theory, the types of speech errors are dysarthria, cerebral palsy, lisp, apraxia and stuttering. However, through the analysis, the writer found only stuttering which are experienced by main character in the ―King‘s Speech‖ movie. The writer also found the treatment to cure speech errors of major character depicted in the movie, they are providing relaxed, not reaching negatively, speaking in slightly and supporting them. The King George VI treated by all of those treatments to reduce his speech errors and together with his psychiatrist presenting some factual information about him. However, the speech errors need to be treated before it becomes something usual occur. Hopefully, this thesis can contribute to education as an analysis of literary work, especially for students of English and Literature Department.

MRI as an Aid to the Differential Diagnosis of Dysarthria of Supra-Nuclear Origin

Abstract: Five cases are presented to compare the relative value of MRI and CT as aids to the differential diagnosis of dysarthria resulting from supra-nuclear lesions. In all five cases the lesions demonstrated by MRI were more extensive than those evidenced by CT. Moreover, although the lesions evident on the MR images for each subject were able to explain and predict the particular speech disorder exhibited, the same was not true for the lesions demonstrated by CT. It was concluded that MRI is more useful as an aid to the differential diagnosis of dysarthria than CT, especially in those cases where the dysarthria is of a mixed-type or the lesions involve structures in the posterior cranial fossa.

The Importance of Dysarthria in Differential Diagnosis: A Case Study

Abstract: Dysarthria results from lesions in the central nervous system, the peripheral nervous system, or both. Therefore, dysarthria is associated with a wide variety of neurological pathologies and presents in a variety of forms. In a given clinical neurological picture, disturbance of speech is usually relegated towards the bottom of the list of symptoms; its presence may be acknowledged but granted little importance for diagnosis and usually for the rehabilitation process as well. The purpose of this chapter is to present a clinical case study illustrating the potential importance of dysarthria in the differential diagnosis of a neurological condition. Our patient was diagnosed with Binswanger’s disease (BD), but the diagnosis did not match the dysarthria or some aspects of the clinical neuropsychological symptomatology. Subsequently, and in part due to the dysarthrie signs, the diagnosis of amyotrophic lateral sclerosis (ALS) was added.

[A 63 year-old man with progressive gait disturbance and dysarthria].

Abstract: We report a 63-year-old man with progressive gait disturbance and dysarthria. The patient was apparently well until the age of 62 (February, 1990) when he noted unsteadiness of gait. Two months later, dysarthria appeared. He was admitted to Juntendo Izunagaoka Hospital on April 23, 1990. Neurologic examination revealed a mentally sound man with normal higher cerebral functions. Cranial nerves were unremarkable except for scanning speech. His gait was ataxic with positive Romberg sign. No motor weakness was noted, however, he had hypotonia and cerebellar ataxia. Deep tendon reflexes were retained and the plantar response was flexor. Pain, touch and vibration senses were diminished in the distal parts of the lower extremities. Laboratory examination revealed a 2.5 cm mass in the left lung field. Cranial MRI revealed a small T1-low and T2-high signal intensity lesion in the left temporal lobe. Abdominal CT scan revealed multiple low density lesions in the liver. His subsequent course was complicated by progressive deterioration in his gait and loss of deep tendon reflexes. He expired on November 24, 1990. The patient was discussed in the neurological CPC and the chief discussant arrived at the conclusion that the patient had anti-Hu associated paraneoplastic encephalomyelitis and sensory neuropathy. Some other participants thought that the patient had carcinomatous cerebellar degeneration. Postmortem examination revealed a 4x4 cm mass lesion involving the left S4-S5 segments. Histologic examination of the tumor was small cell carcinoma. Many metastatic foci were found in the liver. The cerebral hemispheres were unremarkable except for a small wedge-shaped tissue defect in the left temporal lobe which appeared to have been caused by old head trauma which the patient had received. The cerebellar vermis showed slight enlargement of cortical sulci, however, the cerebellar hemispheres appeared unremarkable. Upon histologic examination, marked loss of Purkinje cells was noted, particularly in the cerebellar anterior lobe. The dentate nucleus showed slight cell loss with increase in fat granule cells. The inferior olive was normal. The histologic characteristics were consistent with the pathologic diagnosis of carcinomatous cerebellar degeneration. No evidence of limbic encephalitis was seen. The peripheral nerve was not examined.