Sodium propionate

About: Sodium propionate is a research topic. Over the lifetime, 463 publications have been published within this topic receiving 9451 citations. The topic is also known as: E281 & sodium propionate anhydrous.

Method for preparing antimicrobial salts

Abstract: Novel antimicrobial products and methods of making and using the same are disclosed, whereby the products can be used in the same or greater percentages as conventional microbial growth inhibitors without imparting an off-flavor, taste, color or odor to the products in which they are used. The antimicrobial products are formed by exposing a compound selected from the group consisting of benzoic acid, sodium benzoate, calcium benzoate, potassium benzoate, acetic acid, sodium diacetate, paraben, niacin, calcium acetate, calcium diacetate, citric acid, lactic acid, fumaric acid, sorbic acid, sodium sorbate, calcium sorbate, potassium sorbate, propionic acid, sodium propionate, calcium propionate, potassium propionate and mixtures thereof, to an ammonia gas. In one embodiment, the product is prepared by placing a layer of azodicarbonamide on a substrate and covering the layer with a gas permeable separator. The antimicrobial compound is then added on top of the separator, and the combination is heated to cause the azodicarbonamide to decompose and release ammonia gas to form the final product. In another embodiment, the product is prepared by exposing the antimicrobial compound to an ammonia gas. The ammonia gas reacts with free acids in the antimicrobial compound to convert the free acids into ammonium salts, thereby eliminating off-flavor and off-odor of the resulting antimicrobial product. The antimicrobial products prepared according to the present invention are suitable for use in foodstuffs, sanitation products, cosmetics, pharmaceuticals, and so forth.

Chemical composition, fermentation profile and apparent digestibility of sugarcane silage treated with chemical additives

Abstract: The aim of this study was the indication of an additive for the ensilage of sugarcane (Saccharum officinarum L.). In a laboratory trial, nine treatments were applied (g of additive/kg of fresh forage - FF) to the sugarcane (RB867515), before ensiling in minisilos (15 x 30 cm PVC tubes) during 78 days: untreated; urea (5) + sodium benzoate (0.5); urea (7.5) + benzoate (0.5); urea (5) + benzoate (0.75); urea (7.5) + benzoate (0.75); sodium propionate (1, 2 and 4); calcium hydroxide (10). Urea + benzoate in the lowest doses, propionate in the higher concentration and calcium hydroxide were selected, considering the ethanol content (26.5, 27.2 and 7.4 g/kg DM, respectively), total DM loss (88, 46 and 58 g/kg DM, respectively) and digestibility (541, 496 and 516 g/kg DM, respectively) of the silages. Silages treated with these doses of additives and the untreated silage (80 d of storage) were fed (nine + seven d) to 16 castrated male sheep (Santa Ines) housed in metabolic cages. The silage with calcium hydroxide presented coefficients of apparent digestibility of DM (0.44), of NDF (0.4) and DM intake (20 g/kg live weight) in the higher levels. Calcium hydroxide was superior to propionate and urea + benzoate, considering alcoholic fermentation control and reduction of losses in the silage and the forage’s nutritional value.

Interior wall latex paint

Abstract: The invention discloses interior-wall latex paint. The latex paint comprises 30 to 35% of sodium propionate, 4 to 10% of a phenylpropylene solution, 4 to 9% of a tetraethylammonium hydroxide methanolsolution, 15 to 26% of silica ultrafine powder, 3.0 to 4.0% of trimethylamine hydrochloride, 10 to 14% of a fungicide, 45 to 65% of organic amine and 45 to 55% of deionized water. The interior-wall latex paint disclosed by the invention does not pollute air, and ensures the environmental quality not to be destroyed. Furthermore, the product has relatively strong aging resistance and is easy to clean and turn over.

Photosynthetic bacteria culture simple and convenient propagation culture medium and preparation method thereof

Abstract: The invention discloses a photosynthetic bacteria culture simple and convenient propagation culture medium. Per liter of an aqueous solution comprises the following components by mass: 2.5-3.5 g of sodium propionate, 1.0-2.0 g of ammonium chloride, 2.5-3.5 g of peptone, 8-10 g of sodium chloride, 1.0-1.5 g of sodium bicarbonate. 0.4-0.6 g of potassium dihydrogen phosphate, 0.1-0.3 g of magnesium sulfate, 0.24-0.26 g of sodium hydroxide, 0.03-0.05 g of manganese sulfate, 0.002-0.005 g of ferrous sulfate, and 0.0006-0.001 g of sodium glutamate. The photosynthetic bacteria culture simple and convenient propagation culture medium and the preparation method thereof do not need yeast cream, is low in production cost, and low in equipment requirement. Used water is tap water or well water, production operation is simple and convenient, and capacity of resisting sundry fungus is strong. In addition, photosynthetic bacteria generated by using the photosynthetic bacteria culture medium are highin viable count, and not easy to decay.

Effects of substrates and neuroendocrine factors on expression of PEPCK-C mRNA in neonatal calf hepatocytes in vitro

Abstract: Different concentrations of sodium propionate,sodium pyruvate,insulin,glucagons,and leptin were added respectively into monolayer culture media of primary-cultured calf hepatocytes.After the hepatocytes were cultured for 12h,abundance of PEPCK-C mRNA was detected by semiquantitative RT-PCR in primary-cultured calf hepatocytes.With increase of concentrations of sodium propionate or sodium pyruvate,the abundance of PEPCK-C mRNA in the hepatocytes increased significantly first,whereafter decreased.With increasing levels of insulin,glucagons,and leptin singly,the abundance of PEPCK-C mRNA in the hepatocyte dropped dose-dependently significantly,increased and had no difference,respectively.Sodium propionate and sodium pyruvate could promote liver gluconeogenesis by up-regulating the abundance of PEPCK-C mRNA in the hepatocytes,but the up-regulation was limited.Insulin could dose-(dependently) inhibit liver gluconeogenesis by down-regulating the abundance of PEPCK-C mRNA in the hepatocytes.The effect of glucagon was contrary to that of insulin.Leptin had no directly effect in regulating expression of PEPCK-C mRNA.