Showing papers on "White Muscle Disease published in 1979"
TL;DR: Increased selenium deficiency from the increasing use of home grown feeds as a major constituent of livestock rations may be causally related to the increase of white muscle disease and other seenium responsive diseases in Britain.
Abstract: The reliability of erythrocyte glutathione peroxidase activity as an indicator of selenium status in livestock is discussed. Based on this measurement, a survey is described of the biological selenium status of sheep on each of 329 farms in Britain. Results showed that 47 per cent of these farms were probably unable to provide grazing livestock with sufficient selenium to maintain blood levels greater than 0.075 microgram per ml. Increased selenium deficiency from the increasing use of home grown feeds as a major constituent of livestock rations may be causally related to the increase of white muscle disease and other selenium responsive diseases in Britain.
48 citations
Journal Article•
TL;DR: Although the effects of weight loss from selenium-deficient cattle is less dramatic than death caused by white muscle disease, the economic loss can be severe.
Abstract: Although the effects of weight loss from selenium-deficient cattle is less dramatic than death caused by white muscle disease (also resulting from selenium deficiency), the economic loss can be severe. Treatments with injections of selenium have produced significant weight-gain increases in selenium-deficient areas.
13 citations
Journal Article•
01 Sep 1979-Journal of The South African Veterinary Association-tydskrif Van Die Suid-afrikaanse Veterinere Vereniging
TL;DR: Analysis of the feed showed barely adequate selenium in the maize silage and a deficiency in the soil on which the high lysine maize was grown, suggesting that this deficiency may be more widespread than is presently realised.
Abstract: White muscle disease appeared in lambs born of ewes in the zero grazing group during the course of a comparative fertility trial on ewes on various systems of husbandry. Analysis of the feed showed barely adequate selenium in the maize silage and a deficiency in the soil on which the high lysine maize was grown. Treatment of the lambs with a selenium/vitamin E injection rapidly controlled the condition. This deficiency may be more widespread than is presently realised.
4 citations
Journal Article•
TL;DR: Electron microscopy was used to examine the sekeletal muscles of young cattle with spontaneous enzootic myodystrophy (nutritional myodegeneration due to selenium deficiency, white muscle disease), and there were far-reaching ultrastructural similarities to nutritional myode Generation of sheep.
Abstract: Electron microscopy was used to examine the sekeletal muscles of young cattle, aged between 13 and 24 months, with spontaneous enzootic myodystrophy (nutritional myodegeneration due to selenium deficiency, white muscle disease). The animals had been received from Sumava District, Southern Bohemia, an area known for shortage of selenium. Outbreaks of clinical illness were recorded from them between four and 18 days from the beginning of grazing. Most of the ultrastructural changes included decomposition of myofibrils and hyalinisation of fibres as well as defective fibril synthesis (Z-striation abnormality), some of the latter phenomena recordable even from regenerating fibre. However, minor disorders only were established from the mitochondria, sarcoplasmic reticulum, components of sarcoplasm, and vessels. There were far-reaching ultrastructural similarities to nutritional myodegeneration of sheep. The changes recorded are likely to suggest a specific role played by selenium in the formation and preservation of myofibril proteins.
1 citations