Y
Yoshihiro Sato
Researcher at Kurume University
Publications - 204
Citations - 6846
Yoshihiro Sato is an academic researcher from Kurume University. The author has contributed to research in topics: Osteoporosis & Bone mineral. The author has an hindex of 45, co-authored 202 publications receiving 6595 citations. Previous affiliations of Yoshihiro Sato include Hirosaki University.
Papers
More filters
Journal ArticleDOI
Low-dose vitamin D prevents muscular atrophy and reduces falls and hip fractures in women after stroke: a randomized controlled trial.
TL;DR: Vitamin D may increase muscle strength by improving atrophy of type II muscle fibers, which may lead to decreased falls and hip fractures in patients with long-standing stroke by vitamin D supplementation.
Journal ArticleDOI
Effect of Folate and Mecobalamin on Hip Fractures in Patients With Stroke: A Randomized Controlled Trial
TL;DR: Combined treatment with folate and vitamin B12 is safe and effective in reducing the risk of a hip fracture in elderly patients following stroke in this Japanese population with a high baseline fracture risk.
Journal ArticleDOI
Decreased bone mass and increased bone turnover with valproate therapy in adults with epilepsy
TL;DR: Long-term VPA monotherapy can increase bone Resorption, leading to decreased BMD, implying that increased bone resorption caused the latter.
Journal ArticleDOI
Effect of whole-body vibration exercise and muscle strengthening, balance, and walking exercises on walking ability in the elderly.
Kazuhiro Kawanabe,Akira Kawashima,Issei Sashimoto,Tsuyoshi Takeda,Yoshihiro Sato,Jun Iwamoto +5 more
TL;DR: After the 2-month exercise program, the walking speed, step length, and the maximum standing time on one leg were significantly improved in the WBV exercise plus routine exercises group, while no significant changes were observed in the routine exercises alone group.
Journal ArticleDOI
High prevalence of vitamin D deficiency and reduced bone mass in Parkinson's disease
TL;DR: Vitamin D deficiency due to sunlight deprivation and hypercalcemia induces compensatory hyperparathyroidism, which contributes to reduced BMD in PD patients, particularly those who are functionally dependent, but may be improved by vitamin D supplementation.