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Abdul J. Tajik

Researcher at Mayo Clinic

Publications -  139
Citations -  15926

Abdul J. Tajik is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Mitral valve & Doppler echocardiography. The author has an hindex of 54, co-authored 138 publications receiving 15458 citations. Previous affiliations of Abdul J. Tajik include University of Rochester.

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Clinical Utility of Doppler Echocardiography and Tissue Doppler Imaging in the Estimation of Left Ventricular Filling Pressures A Comparative Simultaneous Doppler-Catheterization Study

TL;DR: The combination of tissue Doppler imaging of the mitral annulus and mitral inflow velocity curves provides better estimates of LV filling pressures than other methods (pulmonary vein, preload reduction), however, accurate prediction of filling pressures for an individual patient requires a stepwise approach incorporating all available data.
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New index of combined systolic and diastolic myocardial performance: a simple and reproducible measure of cardiac function--a study in normals and dilated cardiomyopathy

TL;DR: (ICT+IRT)/ET is a conceptually new, simple and reproducible Doppler index of combined systolic and diastolic myocardial performance in patients with primarymyocardial systols dysfunction and was easily measured, reproducible, and had a narrow range in normals.
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The natural history of idiopathic dilated cardiomyopathy

TL;DR: Between 1960 and 1973, a total of 104 patients at the Mayo Clinic had a diagnosis of idiopathic dilated cardiomyopathy on the basis of clinical and angiographic criteria; these patients were followed up for 6 to 20 years.
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Valve repair improves the outcome of surgery for mitral regurgitation: A multivariate analysis

TL;DR: Valve repair significantly improves postoperative outcome in patients with mitral regurgitation and should be the preferred mode of surgical correction.
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Human Aortic Valve Calcification Is Associated With an Osteoblast Phenotype

TL;DR: These findings support the concept that aortic valve calcification is not a random degenerative process but an active regulated process associated with an osteoblast-like phenotype.