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Achilles K. Papavasiliou
Researcher at Albert Einstein College of Medicine
Publications - 4
Citations - 401
Achilles K. Papavasiliou is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Progenitor cell & Neurosphere. The author has an hindex of 4, co-authored 4 publications receiving 396 citations. Previous affiliations of Achilles K. Papavasiliou include Yeshiva University.
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Bone Morphogenetic Proteins Induce Astroglial Differentiation of Oligodendroglial–Astroglial Progenitor Cells
TL;DR: It is reported that the bone morphogenetic proteins (BMPs), a major subclass of the transforming growth factor β (TGFβ) superfamily, promote the selective, dose-dependent differentiation of O-2As into astrocytes with concurrent suppression of oligodendroglial differentiation.
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Cytokines regulate the cellular phenotype of developing neural lineage species.
Mark F. Mehler,Ronen Marmur,Robert E. Gross,Peter C. Mabie,Ziying Zang,Achilles K. Papavasiliou,John A. Kessler +6 more
TL;DR: Examination of the expression of hematolymphopoietic cytokines and their receptors in brain and neural cultures has confirmed that these epigenetic signals are present at the appropriate development times to mediate their neurotrophic actions.
Journal ArticleDOI
Microglial lineage species are expressed in mammalian epidermal growth factor-generated embryonic neurospheres.
Achilles K. Papavasiliou,Mark F. Mehler,Kostantin Dobrenis,Ronen Marmur,Peter C. Mabie,John A. Kessler +5 more
TL;DR: Observations indicate that microglia are present within the generative zone progenitor cell system, and this system thus represents an important experimental resource to examine the progenitors cell maturation and the origin of the microglial lineage.
Journal ArticleDOI
Paracrine regulation of colony-stimulating factor-1 in medulloblastoma: implications for pathogenesis and therapeutic interventions.
Achilles K. Papavasiliou,Mark F. Mehler,Peter C. Mabie,Ronen Marmur,Song Qingbin,Robert F. Keating,John A. Kessler +6 more
TL;DR: It is suggested that CSF-1 mediates important paracrine interactions between transformed cells and the immune system, resulting in increased growth rate and metastatic potential.