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Adrian M. Medioli

Researcher at University of Newcastle

Publications -  21
Citations -  258

Adrian M. Medioli is an academic researcher from University of Newcastle. The author has contributed to research in topics: Model predictive control & Set (abstract data type). The author has an hindex of 6, co-authored 21 publications receiving 229 citations.

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Emulation-Based Virtual Laboratories: A Low-Cost Alternative to Physical Experiments in Control Engineering Education

TL;DR: The case for emulation-based virtual laboratories in control engineering education is argued and it is demonstrated that such emulation experiments can give students an industrially relevant educational experience at relatively low cost.
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A fundamental control limitation for linear positive systems with application to Type 1 diabetes treatment

TL;DR: A fundamental design trade-off applicable to a class of linear positive systems is presented, which provides a defensible benchmark for comparison of all possible insulin treatment strategies for Type 1 diabetes patients and has potential application in many areas.
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Scenario-based, closed-loop model predictive control with application to emergency vehicle scheduling

TL;DR: In this paper, a closed-loop model predictive control strategy is proposed for fluid deployment of ambulance resources in an emergency vehicle scenario, where disturbances are the dominant feature of the system.

Scenario-based, closed-loop model predictive control with application to emergency vehicle scheduling | NOVA. The University of Newcastle's Digital Repository

TL;DR: This work develops a novel closed-loop model predictive control strategy aimed at this class of problems motivated by the scheduling of emergency vehicles, and motivate the ideas via the problem of fluid deployment of ambulance resources.
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A fundamental control performance limit for a class of positive nonlinear systems

TL;DR: The result provides a fundamental performance standard against which all control policies, including closed loop schemes, can be compared and implications are examined in the context of blood glucose regulation for Type 1 Diabetes.