Aiko Kurisaki-Arakawa

TL;DR: In this article, the authors examined variations of the NAB2-STAT6 fusion gene in 40 cases of SFT using formalin-fixed, paraffin-embedded tissues and secondary genetic alterations of tumor protein p53 (TP53),, platelet-derived growth factor receptor, β polypeptide (PDGFRB), and telomerase reverse transcriptase (TERT) promoters.

TL;DR: In PTCs, telomerase activation by TERT promoter mutation might be more important than alternative lengthening of telomere maintenance by immunohistochemical staining for ATRX and DAXX.

TL;DR: Tests on triple‐negative breast cancers with a high density of tumor‐infiltrating lymphocytes found that MSI‐H tumors were absent in TIL‐high breast cancers, and examination of MMR proteins and PD‐L1 expression may merit further studies to yield predictive information for identifying patients who are likely to benefit from ICI.

TL;DR: A case of dedifferentiated SFT arising within the pelvis of a 70-year-old Japanese woman identifies a possible role of p53 dysfunction in the dedifferentiation process of SFT as reported in other sarcomas.

TL;DR: This case presented with an intramuscular tumor in the region of the gastrocnemius, the first case of deep LGFMS arising in a child younger than 5 years of age and still alive with no evidence of the disease 4 months after diagnosis.