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Ajay K. Singh

Researcher at Brigham and Women's Hospital

Publications -  2123
Citations -  18370

Ajay K. Singh is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 50, co-authored 335 publications receiving 13916 citations. Previous affiliations of Ajay K. Singh include Tufts University & Johns Hopkins University.

Papers
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Synergizing Microbial Enriched Municipal Solid Waste Compost and Mineral Gypsum for Optimizing Rice-Wheat Productivity in Sodic Soils

TL;DR: In this article , a field experiment was conducted in sodic soil to study the performance of microbial enriched municipal solid waste compost (EMSWC) alone and in combination with a reduced dose of gypsum on growth, yield, nutrient uptake and grain quality of rice and wheat.
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Machine Learning Guided Structure Function Predictions Enable In Silico Nanoparticle Screening for Polymeric Gene Delivery.

TL;DR: In this paper , a random forest model was used to predict DNA transfection in vitro based on the chemical structure of the constituent poly(beta-amino ester)s (PBAEs) in a cell line dependent manner.
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Does conservation tillage on partially reclaimed sodic soil restore land degradation by influencing boron dynamics, bioavailability for plant's uptake and nutritional security?

TL;DR: In this paper , a field experiment was initiated for B restoration by incorporating crop residue (CR) into partially reclaimed sodic soil under conservation tillage (zero or reduced tillage) and the results were compared with conventional tillage practices.
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Spectroscopic investigation on the interaction of direct yellow-27 with protein (BSA)

TL;DR: In this paper , the authors investigated the toxicity mechanism of DY-27 interaction with bovine serum albumin (BSA) using multiscale multi-spectroscopic techniques.
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ITK independent development of Th17 responses during hypersensitivity pneumonitis driven lung inflammation

TL;DR: In this article , the authors show that the ability to pharmaceutically target ITK to suppress Th17 responses may be dependent on the type of inflammation, such as Saccharopolyspora rectivirgula (SR) induced hypersensitivity pneumonitis (SR-HP).