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Angela Garcia-Brugger

Researcher at University of Burgundy

Publications -  5
Citations -  971

Angela Garcia-Brugger is an academic researcher from University of Burgundy. The author has contributed to research in topics: Elicitor & Plant defense against herbivory. The author has an hindex of 5, co-authored 5 publications receiving 860 citations. Previous affiliations of Angela Garcia-Brugger include Centre national de la recherche scientifique.

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Journal ArticleDOI

Early signaling events induced by elicitors of plant defenses.

TL;DR: Early signaling events that happen after elicitor perception are reviewed, including reversible protein phosphorylations, changes in the activities of plasma membrane proteins, variations in free calcium concentrations in cytosol and nucleus, and production of nitric oxide and active oxygen species.
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Bacterial rhamnolipids are novel MAMPs conferring resistance to Botrytis cinerea in grapevine.

TL;DR: It is proposed that rhamnolipids are acting as microbe-associated molecular patterns (MAMPs) in grapevine and that the combination of rhamNolipid effects could participate in Grapevine protection against grey mould disease.
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Involvement of the glutamate receptor AtGLR3.3 in plant defense signaling and resistance to Hyaloperonospora arabidopsidis.

TL;DR: Genetic and pharmacological data provide evidence for the involvement of GLRs in elicitor/pathogen-mediated plant defense signaling pathways in Arabidopsis thaliana and some OGs-triggered immune events such as defense gene expression, NO and ROS production are to different extents dependent on AtGLR3.3.
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Involvement of putative glutamate receptors in plant defence signaling and NO production.

TL;DR: An involvement of a putative iGluR in plant defence signaling and NO production is demonstrated for the first time, by mechanisms that show homology with glutamate mode of action in mammals.
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Calcium signatures and signaling in cytosol and organelles of tobacco cells induced by plant defense elicitors.

TL;DR: The results indicate that cryptogein profoundly impacted cell functions at many levels, including organelles, and pharmacological studies indicated that IP(3) should play an important role in Ca(2+) signaling contrarily to cADPR or nitric oxide, which have limited or no effect on [Ca(2+)] variations.