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Arnulf Ferlitsch

Researcher at Medical University of Vienna

Publications -  165
Citations -  5723

Arnulf Ferlitsch is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Cirrhosis & Portal hypertension. The author has an hindex of 37, co-authored 158 publications receiving 4606 citations. Previous affiliations of Arnulf Ferlitsch include University of Vienna & St John of God Health Care.

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Nonselective β blockers increase risk for hepatorenal syndrome and death in patients with cirrhosis and spontaneous bacterial peritonitis.

TL;DR: Among patients with cirrhosis and SBP, NSBBs increase the proportion who are hemodynamically compromised, time of hospitalization, and risks for hepatorenal syndrome and acute kidney injury, and they also reduce transplant-free survival.
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Non-selective betablocker therapy decreases intestinal permeability and serum levels of LBP and IL-6 in patients with cirrhosis.

TL;DR: NSBB treatment ameliorates gastroduodenal/intestinal permeability and reduces bacterial translocation partially independent of their hemodynamic effects on portal pressure, which may contribute to a reduced risk of variceal bleeding.
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Child-Pugh versus MELD score in predicting survival in patients undergoing transjugular intrahepatic portosystemic shunt

TL;DR: Although MELD was the primary predictor of overall survival in multivariate analysis, c statistics showed that both scores can be used for patients undergoing TIPS with equal accuracy, and there seems little reason to replace the well established Child-Pugh score.
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Carvedilol for primary prophylaxis of variceal bleeding in cirrhotic patients with haemodynamic non-response to propranolol

TL;DR: Using carvedilol for primary prophylaxis a substantial proportion of non-responders to propranolol can achieve a haemodynamic response, which is associated with improved outcome with regard to prevention of variceal bleeding, hepatic decompensation and death.
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Sustained virologic response to interferon-free therapies ameliorates HCV-induced portal hypertension

TL;DR: SVR to IFN-free therapies might ameliorate portal hypertension across all BL HVPG strata, however, changes inHVPG seemed to be more heterogeneous among patients with BL HvPG of ⩾16mmHg and a HV PG decrease was less likely in patients with more advanced liver dysfunction.