C
Carmen Cuéllar
Researcher at Complutense University of Madrid
Publications - 99
Citations - 1597
Carmen Cuéllar is an academic researcher from Complutense University of Madrid. The author has contributed to research in topics: Anisakis simplex & Antigen. The author has an hindex of 21, co-authored 97 publications receiving 1467 citations.
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The Anisakis allergy debate: does an evolutionary approach help?
TL;DR: Allergic phenomena share common pathways with the immune response against helminth parasites, as well as putatively crossreacting antibodies, as are used in food allergy, depend on the clinical relevance of specific IgE and deserve careful interpretation in the various forms of A. simplex.
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Anisakis simplex: The high prevalence in Madrid (Spain) and its relation with fish consumption
TL;DR: Positivity was more prevalent among subjects who habitually consumed fresh and possibly undercooked fish than among those who generally consumed frozen fish or boiled or baked fish, and no evidence of cross-reactivity between the ELISA and other allergens was found.
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Recidivous acute urticaria caused by Anisakis simplex
TL;DR: A. simplex was found to be the main cause of acute recidivous urticaria in patients who usually eat fish and are not sensitized to it, and specific immunoglobulin levels against excretory‐secretory antigen were measured by ELISA.
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The Anisakis simplex Ani s 7 major allergen as an indicator of true Anisakis infections
A. M. Anadón,F. Romarís,M. Escalante,Esperanza Rodríguez,Teresa Gárate,Carmen Cuéllar,Florencio M. Ubeira +6 more
TL;DR: The results demonstrate that nAni s 7 is secreted and recognized by the immune system of rats only when the larvae are alive (i.e. during the acute phase of infection), and that this molecule is not present in, or is antigenically different from, Pseudoterranova allergens.
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Gastro-allergic anisakiasis as a consequence of simultaneous primary and secondary immune response.
TL;DR: The allergic IgE‐mediated reaction in the course of gastro‐allergic anisakiasis involves a parallel secondary Th2 type memory response and a primary immunologic stimulation of both Th2 and Th1 lymphocyte subsets against previously unrecognized antigens.