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Chin-Lin Hsu
Researcher at Chung Shan Medical University
Publications - 29
Citations - 838
Chin-Lin Hsu is an academic researcher from Chung Shan Medical University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 14, co-authored 22 publications receiving 699 citations. Previous affiliations of Chin-Lin Hsu include Chung Hwa University of Medical Technology.
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Journal ArticleDOI
Anti-inflammatory Effects of Phenolic Compounds Isolated from the Fruits of Artocarpus heterophyllus
TL;DR: In vitro anti-inflammatory effects of phenolic compounds isolated from the ethyl acetate extracts of the fruits of Artocarpus heterophyllus Lam indicated that artocarpesin may provide a potential therapeutic approach for inflammation-associated disorders.
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Antiobesity and Hypolipidemic Effects of Polyphenol-Rich Longan (Dimocarpus longans Lour.) Flower Water Extract in Hypercaloric-Dietary Rats
Deng-Jye Yang,Yuan-Yen Chang,Chin-Lin Hsu,Cheng-Wei Liu,Yi-Ling Lin,Yu-Huei Lin,Kai-Cheng Liu,Yi-Chen Chen +7 more
TL;DR: Polyphenol-rich LFWE indeed characterizes antiobesity and hypolipidemic effects in vivo, and also increased fecal triglyceride excretions in HCD rats.
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Effects of Polyphenolic Compounds on Tumor Necrosis Factor-α (TNF-α)-Induced Changes of Adipokines and Oxidative Stress in 3T3-L1 Adipocytes
TL;DR: P-coumaric acid, quercetin, and resveratrol demonstrated inhibitions of TNF-α-induced changes in levels of monocyte chemoattractant protein-1, plasminogen activator inhibitor-1 (PAI-1), and intracellular reactive oxygen species (ROS) in 3T3-L1 adipocytes.
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Antioxidant and Anti-Inflammatory Effects of Orthosiphon aristatus and Its Bioactive Compounds
TL;DR: EEOA and its bioactive compound, ursolic acid, suppress LPS-induced NO and PGE(2) production by inhibiting ROS generation, along with reducing expression of iNOS and COX-2 in RAW 264.7 cells.
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Anticancer effects of flavonoid derivatives isolated from Millettia reticulata Benth in SK-Hep-1 human hepatocellular carcinoma cells.
TL;DR: It is demonstrated that genistein (6) induces apoptosis in SK-Hep-1 cells via both Fas- and mitochondria-mediated pathways.