C
Chung-Hung Tsai
Researcher at Chung Shan Medical University
Publications - 55
Citations - 1423
Chung-Hung Tsai is an academic researcher from Chung Shan Medical University. The author has contributed to research in topics: Arecoline & Oral submucous fibrosis. The author has an hindex of 24, co-authored 55 publications receiving 1295 citations.
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Platelet-rich fibrin increases cell attachment, proliferation and collagen-related protein expression of human osteoblasts.
TL;DR: It is suggested that PRF is capable of increasing osteoblast attachment, proliferation and simultaneously upregulating collagen-related protein production and these actions in combination would effectively promote bone regeneration.
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The up-regulation of cyclooxygenase-2 expression in human buccal mucosal fibroblasts by arecoline: a possible role in the pathogenesis of oral submucous fibrosis.
TL;DR: It is suggested that COX-2 expression is significantly up-regulated in OSF tissues from areca quid chewers and arecoline may among other constituents be responsible for the enhanced COX -2 expression in vivo.
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Ginkgo biloba leaves extract (EGb 761) attenuates lipopolysaccharide-induced acute lung injury via inhibition of oxidative stress and NF-κB-dependent matrix metalloproteinase-9 pathway.
TL;DR: With EGb 761 pretreatment, both lipopolysaccharide (LPS)-induced protein leakage and neutrophil infiltration, and LPS-induced inflammatory responses including increased myeloperoxidase (MPO) activity, lipid peroxidation, and metalloproteinase (MMP)-9 activity, were inhibited, suggesting that the protection mechanism of EGb 661 is by inhibition of NFκB activation, possibly via the up-regulation of antioxidative enzymes.
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The upregulation of type I plasminogen activator inhibitor in oral submucous fibrosis.
TL;DR: It is suggested that PAI-1 expression is significantly upregulated in OSF tissues from areca quid chewers, and arecoline may be responsible for the enhanced PAI -1 expression in vivo.
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The Upregulation of Matrix Metalloproteinase-9 in Inflamed Human Dental Pulps
TL;DR: Compared with clinically healthy human pulp and inflamed human pulp tissue specimens, MMP-9 expression was significantly higher in the inflamed pulps than clinically healthy pulps and may play an important role in the pathogenesis of pulpal inflammation.