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David Jiménez

Researcher at University of Texas Health Science Center at San Antonio

Publications -  235
Citations -  9478

David Jiménez is an academic researcher from University of Texas Health Science Center at San Antonio. The author has contributed to research in topics: Pulmonary embolism & Medicine. The author has an hindex of 41, co-authored 189 publications receiving 6931 citations. Previous affiliations of David Jiménez include University of Texas at San Antonio & Wayne State University.

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Endoscopy-assisted early correction of single-suture metopic craniosynostosis: a 19-year experience.

TL;DR: The results presented show excellent and consistent long-term outcomes that are superior to traditional methods and are associated with minimal trauma, blood loss, and anesthetic exposure, and with short surgical times.
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Incidencia de hipertensión pulmonar tromboembólica crónica sintomática y asintomática

TL;DR: La asociacion entre las recurrencias tromboembolicas y the HPTEC sugiere la necesidad de optimizar la duracion y the intensidad del tratamiento anticoagulante en los pacientes con TEP.
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Propofol for Sedation and Control of Intracranial Pressure in Children

TL;DR: The authors present 2 children with increased ICP in whom prop ofol was used to provide sedation and control ICP and the use of propofol in this setting and its possible applications are discussed.
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Exercise pre-conditioning reduces brain inflammation in stroke via tumor necrosis factor-α, extracellular signal-regulated kinase 1/2 and matrix metalloproteinase-9 activity

TL;DR: It is suggested that exercise-induced up-regulation of tumor necrosis factor-α before stroke and extracellular signal-regulated kinase 1/2 phosphorylation play a role in decreasing brain inflammation by regulating matrix metalloproteinase-9 activity.
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Physicians' management approach to an incidental pulmonary embolism: an international survey.

TL;DR: Lipoprotein (a) levels as an independent risk factor for venous thromboembolism in adults: a meta-analysis and genetic evidence that lipoprotein(a) associates with atherosclerotic stenosis rather than venousThrombosis.