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David M. Harrild

Researcher at Boston Children's Hospital

Publications -  65
Citations -  1573

David M. Harrild is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Tetralogy of Fallot & Medicine. The author has an hindex of 18, co-authored 54 publications receiving 1226 citations. Previous affiliations of David M. Harrild include Duke University & Harvard University.

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Pulmonary Valve Replacement in Tetralogy of Fallot Impact on Survival and Ventricular Tachycardia

TL;DR: Late PVR for symptomatic pulmonary regurgitation/RV dilation did not reduce the incidence of VT or death in TOF after PVR and the hypothesis that PVR leads to improvement in these outcomes was tested.
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Autologous mitochondrial transplantation for dysfunction after ischemia-reperfusion injury

TL;DR: Intramyocardial injection of autologous mitochondria with a tuberculin syringe with the aim of establishing mitochondrial isolation and usage and the results confirmed the ability of mitochondria to be isolated and reprogrammed for use in cardiology.
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Long-Term Pulmonary Regurgitation Following Balloon Valvuloplasty for Pulmonary Stenosis: Risk Factors and Relationship to Exercise Capacity and Ventricular Volume and Function

TL;DR: In this paper, the authors examined the prevalence and predictors of pulmonary regurgitation following balloon dilation for pulmonary stenosis and investigated its impact on ventricular volume and function, and exercise tolerance.
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Comparison of cardiac MRI tissue tracking and myocardial tagging for assessment of regional ventricular strain

TL;DR: The TT approach does not add to CMR examination time and may be a useful tool for the assessment of ventricular synchrony and Measurements of segmental time to peak strain by TT and MT were in close agreement; agreement for the magnitude of peak segmental strain was more modest.
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Circumferential and longitudinal ventricular strain in the normal human fetus.

TL;DR: This is the first study to report normal fetal LV circumferential strain in a normal fetal population, and these data may be useful as a reference for assessing fetal cardiac function.