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Dong Ryeol Ryu

Researcher at Ewha Womans University

Publications -  179
Citations -  2863

Dong Ryeol Ryu is an academic researcher from Ewha Womans University. The author has contributed to research in topics: Kidney disease & Hemodialysis. The author has an hindex of 26, co-authored 166 publications receiving 2238 citations. Previous affiliations of Dong Ryeol Ryu include University of California, Los Angeles & Beckman Research Institute.

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Podocyte biology in diabetic nephropathy

TL;DR: This review explores some of the structural and functional changes of podocytes under diabetic conditions and their role in the development and progression of diabetic nephropathy.
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Changes in causative organisms and their antimicrobial susceptibilities in CAPD peritonitis: a single center's experience over one decade.

TL;DR: The findings suggest that it is necessary to prepare new center-based guidelines for the initial empirical treatment of CAPD peritonitis by examining the changes of causative organisms and their susceptibilities to antimicrobial agents over the past 10 years.
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Activation of the renin-angiotensin system within podocytes in diabetes.

TL;DR: It is suggested that increased AGT, an attendant increase in AII and increased AII type 1 receptor in podocytes experiencing diabetic conditions play an important role in the pathogenesis of diabetic nephropathy.
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A population-based approach indicates an overall higher patient mortality with peritoneal dialysis compared to hemodialysis in Korea

TL;DR: While the overall mortality rate was higher in incident PD patients, mortality rates of some incident PD and HD patients were comparable in Korea, and the survival rate of PD patients was comparable to that of HD patients.
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Sirt1-hypoxia-inducible factor-1α interaction is a key mediator of tubulointerstitial damage in the aged kidney.

TL;DR: The data suggest that Sirt1‐induced deacetylation of HIF‐1α may have protective effects against tubulointerstitial damage in aged kidney and confirmed that chronic activation of Hif‐1 α promoted apoptosis and fibrosis, using tubular cell‐specific HIF-1α transgenic mice.