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Eiichi Abe

Researcher at Iwate Medical University

Publications -  16
Citations -  173

Eiichi Abe is an academic researcher from Iwate Medical University. The author has contributed to research in topics: Monoamine neurotransmitter & Acetylcholine. The author has an hindex of 8, co-authored 16 publications receiving 171 citations.

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Rapid and simultaneous assay of monoamine neurotransmitters and their metabolites in discrete brain areas of mice by HPLC with Coulometric detection

TL;DR: The present procedure was applied to measure the target substrates in several discrete brain areas in mice to enable simultaneous assay of the three monoamine neurotransmitters, norepinephrine, dopamine, and serotonin, and four respective metabolites in brain tissue.
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A rapid assay for neurotransmitter amino acids, aspartate, glutamate, glycine, taurine and γ-aminobutyric acid in the brain by high-performance liquid chromatography with electrochemical detection

TL;DR: A very rapid and simple chromatographic method using high-performance liquid chromatography with electrochemical detection in combination with o-phthalaldehyde derivatization to measure amino acid neurotransmitter levels in several discrete brain regions of mice treated with and without electroconvulsive shock is described.
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MKC-231, a choline uptake enhancer, ameliorates working memory deficits and decreased hippocampal acetylcholine induced by ethylcholine aziridinium ion in mice

TL;DR: Results indicate that MKC-231 improved the AF64A-induced working memory deficit and hippocampal ACh depletion, probably by recovering reduced high-affinity choline uptake and ACh release.
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Effects of nefiracetam on deficits in active avoidance response and hippocampal cholinergic and monoaminergic dysfunctions induced by AF64A in mice.

TL;DR: The results indicate that the effects of nefiracetam on AF64A-induced behavioural deficits are probably due to its ability to facilitate both cholinergic and monoaminergic neurotransmitter systems.
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Effects of 24-Hr fasting on methamphetamine- and apomorphine-induced locomotor activities, and on monoamine metabolism in mouse corpus striatum and nucleus accumbens

TL;DR: The results suggest that the locomotor activity in fasting mice may be increased by a change in the sensitivity of dopaminergic neurons in the corpus striatum.