Ellen C. Peters

TL;DR: The growth retardation was greatest when women were underweight pregravid and had a low pregnancy weight gain, when they were hypertensive, or when the work required standing, and the frequency of large placental infarcts progressively increased when women continued stand-up work into late gestation.

TL;DR: In the victims of cerebral palsy, characteristic consequences of birth asphyxia were more often the result of nonasphyxial disorders, and these included meconium in the amniotic fluid, low 10-minute Apgar scores, neonatal apnea spells, seizures, persisting neurologic abnormalities, and slow head growth after birth.

TL;DR: Maternal gestational weight gains were suboptimal and the involved neonates had a pattern of growth retardation characteristic of undernutrition, and mother's race, socioeconomic status, age, short stature, and number of prior unsuccessful pregnancies lost their positive association with the fatal infections when mothers made more than nine clinic visits.

TL;DR: All of these findings were the same whether neurologic abnormalities were absent or present, suggesting that the same factors were sometimes Involved in the genesis of cognitive impairments and Neurologic abnormalities.

TL;DR: Pregnancy, perinatal, and subsequent developmental data for 19,117 children suggested that the same factors were sometimes involved in the genesis of cognitive impairments and neurologic abnormalities, and antenatal disorders and conditions correlated with low IQ values.