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Emilia Kaivosoja

Researcher at Aalto University

Publications -  24
Citations -  621

Emilia Kaivosoja is an academic researcher from Aalto University. The author has contributed to research in topics: Thin film & Actin cytoskeleton. The author has an hindex of 14, co-authored 24 publications receiving 558 citations. Previous affiliations of Emilia Kaivosoja include New York University & University of Helsinki.

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Circadian timekeeping is disturbed in rheumatoid arthritis at molecular level.

TL;DR: The molecular machinery controlling the circadian rhythm is disturbed in RA patients, and ARNTL2 and NPAS2 appeared to be the most affected clock genes in human immune-inflammatory conditions.
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Chemical and physical properties of regenerative medicine materials controlling stem cell fate

TL;DR: This review focuses on the stem cell–extracellular matrix interactions by summarizing the observations of the effects of material variables (such as overall architecture, surface topography, charge, ζ-potential, surface energy, and elastic modulus) on thestem cell fate.
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Adhesion, spreading and osteogenic differentiation of mesenchymal stem cells cultured on micropatterned amorphous diamond, titanium, tantalum and chromium coatings on silicon

TL;DR: It was hypothesized that human mesenchymal stromal cell (hMSC) can be guided by patterned and plain amorphous diamond (AD), titanium (Ti), tantalum (Ta) and chromium (Cr) coatings, produced on silicon wafer using physical vapour deposition and photolithography.
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Formation and retention of staphylococcal biofilms on DLC and its hybrids compared to metals used as biomaterials.

TL;DR: Ability to resist biofilm formation and attachment could not be explained by only one factor, but it seems to be related to a combination of various properties, with electrokinetic streaming potential and protein coating being particularly important for its outcome.
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Microbial antigens mediate HLA-B27 diseases via TLRs.

TL;DR: It is proposed that the urogenital organs form a source of damage-associated molecular patterns (DAMPs) from microbes or endogenous alarmins, such as uric acid, released from necrotic cells or urate deposits, and placed in the pathogenic centre stage.