Fabiola Terzi

TL;DR: It is concluded that a salt-losing syndrome with tubular resistance to aldosterone can occur during pyelonephritis in young infants with congenital UT malformation, that the risk diminishes considerably or disappears after 3 months of age and that in the absence ofUT malformation pyel onephritis does not cause acute sodium loss of clinical relevance.

TL;DR: Tube feeding may be an effective therapeutic option for overcoming malnutrition when chronic renal failure is associated with persistent anorexia, and growth retardation can be opposed and catch-up growth obtained.

TL;DR: The data show that comprehensive monitoring and suitable treatment must be performed in order to prevent growth retardation at any GFR level and that growth velocity may be normal regardless of statural and bone maturation delay and the degree of renal insufficiency.

TL;DR: Monitoring of serum electrolyte equilibrium and plasma aldosterone concentrations in infants who had severe obstructive uropathy or grade 5 vesico-ureteral reflux concluded that infants undergoing surgical correction of uropathies may require a high sodium intake to maintain electrolyte balance and adequate growth.

TL;DR: LPD induced a decrease in renal plasma flow (RPF) without GFR variation; this finding was annulled when LPD was supplemented either with AAmix or EAKa, suggesting that EAKA used as supplement of LPD in chronic renal disease could counterbalance the low-protein induced reduced glomerular hemodynamic load.