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Forrest C. Bennett

Researcher at University of Washington

Publications -  63
Citations -  3434

Forrest C. Bennett is an academic researcher from University of Washington. The author has contributed to research in topics: Low birth weight & Gestational age. The author has an hindex of 30, co-authored 63 publications receiving 3380 citations. Previous affiliations of Forrest C. Bennett include University of California, San Diego.

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Preventive interventions with low birth weight premature infants: an evaluation of their success.

TL;DR: Conclusions from the studies reviewed indicate that intervention programs designed for LBW preterms have had only modest success in altering neurodevelopmental outcomes, although parent-child interaction has often been facilitated.
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Early intervention for low-birth-weight premature infants: what can we achieve?

TL;DR: Results from all studies suggest that preventive developmental interventions for LBW, premature infants have thus far demonstrated only modest success, and future research endeavours should investigate the type, duration, critical age onset, and intensity of the intervention.
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Pediatricians' Perceptions of the Effectiveness of Early Intervention for At-Risk and Handicapped Children

TL;DR: A detailed question-and-answer set was developed by as mentioned in this paper to evaluate pediatricians' perceptions of the effectiveness of early intervention for handicapped and at-risk children, all board-certified pediatricians in the state of Ohio were surveyed.
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The Uncertainty of Preadolescents’ Chronic Health Conditions and Family Distress:

TL;DR: In this paper, the authors identify the relationship between the degree of uncertainty in the preadolescent's chronic condition and family distress and find that unpredictability of symptoms was significantly associated with greater emotional strain for mothers and greater family/social disruption for fathers.
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Follow-up of infants receiving cranial ultrasound for intracranial hemorrhage.

TL;DR: Only two thirds of children without ICH had a completely normal outcome, reinforcing the concept that factors other than ICH alone contribute to neurodevelopmental morbidity in this population.