G
Gail A. Cresci
Researcher at Cleveland Clinic
Publications - 34
Citations - 2793
Gail A. Cresci is an academic researcher from Cleveland Clinic. The author has contributed to research in topics: Gut flora & Butyrate. The author has an hindex of 13, co-authored 34 publications receiving 2399 citations. Previous affiliations of Gail A. Cresci include Cleveland Clinic Lerner Research Institute & Cleveland Clinic Lerner College of Medicine.
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Journal ArticleDOI
Guidelines for the Provision and Assessment of Nutrition Support Therapy in the Adult Critically Ill Patient
Stephen A. McClave,Robert G. Martindale,Vincent W. Vanek,Mary McCarthy,Pamela R. Roberts,Beth Taylor,Juan B. Ochoa,Lena Napolitano,Gail A. Cresci +8 more
Journal ArticleDOI
Gut Microbiome: What We Do and Don't Know.
Gail A. Cresci,Emmy Bawden +1 more
TL;DR: This article reviews what is currently known regarding various influences on the gut microbiome and is meant to encourage the reader to further explore the unknown.
Journal ArticleDOI
Linking Pathogenic Mechanisms of Alcoholic Liver Disease With Clinical Phenotypes
TL;DR: The mechanisms by which alcohol injures hepatocytes and the response of hepatic sinusoidal cells to alcohol-induced injury are reviewed and how recent insights into the pathogenesis of ALD will affect the treatment and management of patients are discussed.
Journal ArticleDOI
Prophylactic tributyrin treatment mitigates chronic-binge ethanol-induced intestinal barrier and liver injury.
Gail A. Cresci,Bryan Glueck,Megan R. McMullen,Wei Xin,Daniella Allende,Laura E. Nagy,Laura E. Nagy +6 more
TL;DR: This study aimed to determine whether prophylactic tributyrin could protect the intestinal barrier and liver in mice during combined chronic chronic chronic‐binge ethanol exposure.
Journal ArticleDOI
Tributyrin supplementation protects mice from acute ethanol-induced gut injury.
TL;DR: Protection of the intestine with tributyrin supplementation was accompanied by mitigation of EtOH-induced increases in aspartate aminotransferase and inflammatory measures in the short-term and acute EtOH exposure protocols, but not after chronic EtOH feeding.