Showing papers by "Gert Pfurtscheller published in 1988"
TL;DR: Single-lead EEG data referred to one ear were recorded during voluntary finger movements, and transverse bipolar, source and common average reference derivations and the laplacian operator were calculated, and ERD maps are computed.
95 citations
TL;DR: The results suggest that lateral eye movements indicate individual differences in hemispheric EEG-asymmetries under certain experimental conditions (task, segment of EEG-analysis, site, frequency band).
13 citations
TL;DR: The parallel improvement of rCBF and EEG in brain regions surrounding chronic infarcts in 3 patients was interpreted as functional improvement as a consequence of nimodipine-induced normalization of peri-infarct hypoperfusion, i.e. reversal of flow-dependent neuronal silence and/or dysfunction.
Abstract: In a series of 13 patients with cerebrovascular occlusive disease regional cerebral blood flow (rCBF) measurements (two-dimensional intravenous 133Xe clearance method) and quantitative EEG analysis (sensorimotor rhythms) as well as electronic measurement of handforce were performed before and during intravenous infusion of 1 microgram kg/min of one of the lipophilic dihydropyridine calcium channel blocker nimodipine (Nimotop). The aim of the study was to test the hypothesis of the existence of hypoperfusion (ischaemic penumbra) in the surroundings of chronic cerebral infarcts. All 3 parameters improved in one patient. Sensorimotor rhythms increased in 5 patients, rCBF in 3. EEG and rCBF improved in 2 patients. In 3 instances, a redistribution of rCBF in favour of the peri-infarct zone was noted (significant increase of rCBF from 35 +/- 2 SEM to 53 +/- 4 ml/100 g/min (p less than 0.01), whereas rCBF fell from 61 +/- 5 to 46 +/- 2 ml/100 g/min on a collimator remote from the infarct but in the infarcted hemisphere. The parallel improvement of rCBF and EEG in brain regions surrounding chronic infarcts in 3 patients was interpreted as functional improvement as a consequence of nimodipine-induced normalization of peri-infarct hypoperfusion, i.e. reversal of flow-dependent neuronal silence and/or dysfunction.
7 citations
3 citations
TL;DR: Using miniature earphones and sound tubes no increase of ICP was noted in any patient, and hence these can be recommended for stimulating BAEP in case of increased ICP.
Abstract: Ten measurements of intracranial pressure (ICP) (ventricular n = 5, epidural n = 3) in 8 patients (3 after aneurysm surgery, 5 with head trauma) were performed before and after application of conventional headphones for stimulating brainstem auditory evoked potentials (BAEP). The effects of miniature earphones and sound tubes on ICP were also studied. In 7 of 10 measurements after application of headphones a reversible increase of ICP (mean 26 +/- 19% in patients with ICP greater than 10 mmHg was recorded; in 3 patients (ICP less than or equal to 10 mgHg) no changes of ICP were seen. Using miniature earphones and sound tubes no increase of ICP was noted in any patient, and hence these can be recommended for stimulating BAEP in case of increased ICP.
2 citations
Journal Article•
TL;DR: Comparison of the electrophysiological and computer tomography findings showed a laterally correlated change in the occipital VEP: contralateral to the injured hemisphere, the VEP was dominant, ipsilateral it was reduced or extinguished.
Abstract: Visual evoked potentials (VEPs) were examined as an integrated parameter in a multimodal EP study of brain bioelectrical activity in 33 comatose patients (Glasgow Coma Score 3-6) after craniocerebral trauma. Recordings were performed not only from the occipital (01-02) but also from the central (Cz-C3, Cz-A1, Cz-C4) and frontal (F3-F4) areas. The results show an attenuation of the vertex VEP (SNR 3.2 +/- 1.7) and an increase in the occipital VEP (SNR 4.8 +/- 2.3) as compared to the control group (SNR vertex 7.0 +/- 3.0/SNR occipital 3.9 +/- 2.0). In addition, 16 of the comatose patients showed a very considerable loss of the normally polyphasic character of the occipital VEP in favour of a potential that looked almost monophasic; this was either left or right dominant, and was recorded as a negative or positive potential, respectively, in bipolar recording (01-02). Comparison of the electrophysiological and computer tomography findings showed a laterally correlated change in the occipital VEP: contralateral to the injured hemisphere, the VEP was dominant, ipsilateral it was reduced or extinguished.
2 citations