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Gianluigi Fabbriciani

Researcher at University of Perugia

Publications -  24
Citations -  481

Gianluigi Fabbriciani is an academic researcher from University of Perugia. The author has contributed to research in topics: Osteoporosis & Osteomalacia. The author has an hindex of 12, co-authored 24 publications receiving 435 citations. Previous affiliations of Gianluigi Fabbriciani include University of Milan.

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High weight or body mass index increase the risk of vertebral fractures in postmenopausal osteoporotic women.

TL;DR: Among postmenopausal women with osteoporosis, body weight and BMI are associated with a higher likelihood of having a vertebral fracture, irrespective of the positive association between weight and BMD.
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Bone mineral density in haemophilia patients. A meta-analysis

TL;DR: This meta-analysis confirms the association between severe haemophilia and low BMD and future studies should investigate fracture rates and interventions to prevent bone loss in persons with haemophile.
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Association between circulating osteoprogenitor cell numbers and bone mineral density in postmenopausal osteoporosis.

TL;DR: It is found that alkaline-phosphatase-positive (AP+) cells are the lacking cells in osteoporosis, whose reduction is related to bone loss, and the increased number of alkaline phosphatase/CD34-positive cells may reflect the reactive bone marrow contribution to bone formation.
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Osteoanabolic therapy: a non-surgical option of treatment for Kümmell’s disease?

TL;DR: The case of an 81-year-old osteoporotic woman presented with a 1-year history of persistent low back pain onset after a trauma and treatment with teriparatide is described, which suggests osteoanabolic treatment may be used in patients with Kümmell’s disease to enhance vertebral fracture's healing and contribute to back pain relief.
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Circulating immature osteoprogenitor cells and arterial stiffening in postmenopausal osteoporosis.

TL;DR: In women with postmenopausal osteoporosis an increased availability of circulating osteoprogenitor cells has a detrimental influence on arterial compliance, which may in part explain the association between osteoporeosis and arterial stiffening.