G
Griselda A. Pargament
Researcher at University of Buenos Aires
Publications - 7
Citations - 467
Griselda A. Pargament is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Nitric oxide & Superoxide dismutase. The author has an hindex of 5, co-authored 7 publications receiving 454 citations.
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Journal ArticleDOI
Kinetics of nitric oxide and hydrogen peroxide production and formation of peroxynitrite during the respiratory burst of human neutrophils
Maria Cecilia Carreras,Griselda A. Pargament,Sergio D. Catz,Juan José Poderoso,Alberto Boveris +4 more
TL;DR: The results indicate that .NO release is part of the integrated response of stimulated human neutrophils and that, in these cells, kinetics of ″NO and O2 .− release favour the formation of other oxidants like peroxynitrite.
Journal ArticleDOI
Neutrophil function, nitric oxide, and blood oxidative stress in Parkinson's disease
Emilia Gatto,Maria Cecilia Carreras,Griselda A. Pargament,Natalia A. Riobo,Claudia Reides,Marisa G. Repetto,Manuel Fernandez Pardal,Susana Llesuy,Juan José Poderoso +8 more
TL;DR: It is suggested that neurophils express a primary alteration of ·NO release in PD patients, whereas H2O2 and oxidativestress parameters are more probably related to the evolution of PD or to effects of treatment with L‐dopa.
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Decreased production of nitric oxide by human neutrophils during septic multiple organ dysfunction syndrome. Comparison with endotoxin and cytokine effects on normal cells.
Maria Cecilia Carreras,Sergio D. Catz,Griselda A. Pargament,C G Del Bosco,Juan José Poderoso +4 more
TL;DR: In this article, a study was conducted to determine nitric oxide and superoxide anion release by neutrophils in the septic multiple organ dysfunction syndrome (MODS) and to compare them with the response of normal cells to lipopolysaccharide (LPS) and cytokines.
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Effects of respiratory burst inhibitors on nitric oxide production by human neutrophils
TL;DR: The results suggest that the activation of the tyrosine kinase pathway in stimulated human neutrophils controls positively O2.- and H2O2 generation and simultaneously maintains .NO production in low levels.
Journal ArticleDOI
Circulating plasma factors in Parkinson’s disease enhance nitric oxide release of normal human neutrophils
Emilia Gatto,Natalia A. Riobo,Maria Cecilia Carreras,Francisco J. Schopfer,Griselda A. Pargament,Juan José Poderoso +5 more
TL;DR: The data suggest that an overproduction of *NO related to plasma circulating factors, already detected at initial stages of the disease, participates in the pathophysiology of Parkinson's disease.