H
Hakim Celik
Researcher at Harran University
Publications - 161
Citations - 3993
Hakim Celik is an academic researcher from Harran University. The author has contributed to research in topics: Oxidative stress & Paraoxonase. The author has an hindex of 34, co-authored 150 publications receiving 3597 citations. Previous affiliations of Hakim Celik include University of Gaziantep.
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Total oxidative/anti-oxidative status and relation to bone mineral density in osteoporosis.
TL;DR: The results of this study indicated that increased osteoclastic activity and decreased osteoblastic activity may be associated with an imbalance between oxidant and antioxidant status in postmenopausal osteoporosis and supplementation of antioxidant-enriched diet to the therapy might shed light on the development of novel therapeutic strategies for osteopOrosis.
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Increased oxidative stress and its relation with collagen metabolism in knee osteoarthritis
TL;DR: The present results indicate that the oxidant parameters increased and antioxidant parameters decreased in patients with osteoarthritis; therefore, these patients may be exposed to a potent oxidative stress.
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Increased oxidative stress associated with the severity of the liver disease in various forms of hepatitis B virus infection.
TL;DR: Oxidative stress occurs secondarily to increased total lipid peroxidation and inadequate total antioxidant response and is related to severity of the disease and replication status of virus in hepatitis B infection.
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Effect of pistachio diet on lipid parameters, endothelial function, inflammation, and oxidative status: a prospective study.
Ibrahim Sari,Yasemin Baltaci,Cahit Bagci,Vedat Davutoglu,Ozcan Erel,Hakim Celik,Orhan Ozer,Nur Aksoy,Mehmet Aksoy +8 more
TL;DR: In this paper, the effect of the Antep pistachio (Pistacia vera L.) on blood glucose, lipid parameters, endothelial function, inflammation, and oxidation in healthy young men living in a controlled environment was investigated.
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Increased DNA damage and oxidative stress in patients with cutaneous leishmaniasis.
TL;DR: Findings support the notion that ROS and RNS produced by the organism as a defense strategy may amplify the leishmanicidal activity in patients with CL and patients must be treated urgently to counteract the oxidative DNA damage.