H
Hans Glaumann
Researcher at Karolinska University Hospital
Publications - 170
Citations - 7077
Hans Glaumann is an academic researcher from Karolinska University Hospital. The author has contributed to research in topics: Hepatitis & Cirrhosis. The author has an hindex of 46, co-authored 170 publications receiving 6830 citations. Previous affiliations of Hans Glaumann include Karolinska Institutet.
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Journal ArticleDOI
Decreased survival of subjects with elevated liver function tests during a 28‐year follow‐up
Cecilia Söderberg,Per Stål,Per Stål,Johan Askling,Hans Glaumann,Greger Lindberg,Joel Marmur,Rolf Hultcrantz,Rolf Hultcrantz +8 more
TL;DR: Patients with NASH are at increased risk of death compared with the general population, and liver disease is the third most common cause of death among patients with NAFLD.
Journal ArticleDOI
Natural history of liver disease in cystic fibrosis.
TL;DR: Liver disease was no more frequent at the end of the study period although the median age of the patient population had increased, and modern treatment might positively influence liver disease because it seemed less common, less progressive, and less serious than previously reported.
Journal ArticleDOI
Liver Investigation in 149 Asymptomatic Patients with Moderately Elevated Activities of Serum: Aminotransferases
TL;DR: Diagnosis in this group of patients with persistently raised serum levels of aminotransferases cannot be made without liver biopsy, and blood tests were of little value for distinguishing among different diagnostic groups.
Journal ArticleDOI
Risk factors and clinical presentation of hepatobiliary carcinoma in patients with primary sclerosing cholangitis: A case‐control study
TL;DR: Clinical and biochemical presentation of PSC patients with and without hepatobiliary carcinoma did not differ during the year before cancer diagnosis/Ltx, and smoking seems to be a risk factor for developing hepatobility carcinoma in patients with PSC.
Book ChapterDOI
Mechanisms of Intralysosomal Degradation with Special Reference to Autophagocytosis and Heterophagocytosis of Cell Organelles
TL;DR: The chapter discusses an experimental model in which intracellular organelles are introduced into the lysosomal apparatus of Kupffer cells by means of heterophagy, and the study of the heterophagic model has advantages over induced autophagy because the degradation of each membrane constituent or cell organelle can be separately studied.