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Hans U. Häring

Researcher at University of Tübingen

Publications -  84
Citations -  6992

Hans U. Häring is an academic researcher from University of Tübingen. The author has contributed to research in topics: Insulin & Insulin resistance. The author has an hindex of 45, co-authored 84 publications receiving 6593 citations. Previous affiliations of Hans U. Häring include Dalian Institute of Chemical Physics.

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Plasma Adiponectin Concentrations Predict Insulin Sensitivity of Both Glucose and Lipid Metabolism

TL;DR: In nondiabetic individuals, high adiponectin predicts increased insulin sensitivity, independent of low body fat mass and affects not only insulin-stimulated glucose disposal but also lipoprotein metabolism and insulin-mediated suppression of postprandial FFA release.
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Endothelial Dysfunction Is Detectable in Young Normotensive First-Degree Relatives of Subjects With Type 2 Diabetes in Association With Insulin Resistance

TL;DR: There is a significant association between ED and insulin resistance in young FDRs of DM subjects independent of the classic cardiovascular risk factors.
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Association of the T-G Polymorphism in Adiponectin (Exon 2) With Obesity and Insulin Sensitivity: Interaction With Family History of Type 2 Diabetes

TL;DR: In individuals without familial predisposition for type 2 diabetes, the adiponectin polymorphism may mildly increase the obesity risk (and secondarily insulin resistance) in contrast, in individuals who are already burdened by other genetic factors, this small effect may be very hard to detect.
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Effects of intravenous and dietary lipid challenge on intramyocellular lipid content and the relation with insulin sensitivity in humans.

TL;DR: The data from the lipid infusion protocol suggest a functional relationship between IMCL levels and insulin sensitivity and suggest similar effects could be induced by a high-fat diet, thereby underlining the physiological relevance of these observations.
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The Peroxisome Proliferator-Activated Receptor-γ2 Pro12Ala Polymorphism

TL;DR: The Pro12Ala polymorphism in PPAR-γ2 represents the first genetic variant with a broad impact on the risk of common type 2 diabetes and the precise understanding of its mechanism may lead to novel diagnostic, preventive, and therapeutic approaches for improving the management of type 2abetes.