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Hao-Cheng Lin

Researcher at Peking University

Publications -  27
Citations -  196

Hao-Cheng Lin is an academic researcher from Peking University. The author has contributed to research in topics: Medicine & Azoospermia. The author has an hindex of 6, co-authored 17 publications receiving 112 citations.

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In vitro dentin barrier cytotoxicity testing of some dental restorative materials

TL;DR: The tested cytotoxicity of materials applied on 300-, 500-, and 1000-μm dentin disks indicates that the clinical use of the test materials (excepting self-etching adhesives) in deep cavities poses a potential risk of damage to the pulp tissues to an extent, depending on the thickness of the remaining dentin.
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Ambient ozone pollution is associated with decreased semen quality: longitudinal analysis of 8945 semen samples from 2015 to 2018 and during pollution-control period in Beijing, China.

TL;DR: This study suggested that only O3 exposure was harmful to semen quality and should not be neglected during pollution control operation, and other five pollutants' concentrations decreased compared to those in previous years.
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Altered microRNA profiles of testicular biopsies from patients with nonobstructive azoospermia.

TL;DR: A combination of two miRNAs (miR-10b-3p and miR-34b-5p) that could serve as a predictive biomarker of azoospermia are identified that could be useful for predicting and diagnosing the presence of testicular sperm in individuals with azoOSpermia.
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Nanotechnology-assisted adipose-derived stem cell (ADSC) therapy for erectile dysfunction of cavernous nerve injury: In vivo cell tracking, optimized injection dosage, and functional evaluation

TL;DR: It is demonstrated that nanotechnology can be used to further improve the effect of small dosage of ADSCs to improve erectile function and is related to the regeneration of the smooth muscle, endothelium, and nerve tissues.
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Sodium-Hydrogen-Exchanger expression in human sperm and its relationship with semen parameters

TL;DR: The expression pattern of sNHE suggested that this protein may be involved in the regulation of sperm motility, and aberration of its expression in sperm may contribute to the pathogenesis of asthenozoospermia.