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Helge Skulstad

Researcher at Oslo University Hospital

Publications -  110
Citations -  4068

Helge Skulstad is an academic researcher from Oslo University Hospital. The author has contributed to research in topics: Ejection fraction & Heart failure. The author has an hindex of 28, co-authored 97 publications receiving 3154 citations. Previous affiliations of Helge Skulstad include Akershus University Hospital & University of Oslo.

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Journal ArticleDOI

Detection of ischemia and new insight into left ventricular physiology by strain Doppler and tissue velocity imaging: assessment during coronary bypass operation of the beating heart.

Helge Skulstad, +7 more
- 01 Dec 2004 - 
TL;DR: Strain by Doppler is a sensitive means for detecting myocardial ischemia, also capable of correctly localizing the ischemIA, as opposed to tissue velocity assessment and velocity measurements provided new physiological information.
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The role of echocardiography in quantification of left ventricular dyssynchrony: state of the art and future directions

Otto A. Smiseth, +2 more
- 01 Jan 2012 - 
TL;DR: It is proposed to use onset septal shortening during pre-ejection for timing of earliest left ventricular (LV) electrical activation as well as time of active force generation as defined by LV pressure-strain loops as a method to identify electrical dyssynchrony.
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Postsystolic shortening is a strong predictor of recovery of systolic function in patients with non-ST-elevation myocardial infarction

Christian Eek, +7 more
- 01 Jul 2011 - 
TL;DR: Post-systolic shortening is associated with improved myocardial function after revascularization in patients with acute MI, it predicts long-term systolic function, and provides information on the potential benefit of the procedure.
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Regional myocardial work by strain Doppler echocardiography and LV pressure: a new method for quantifying myocardial function.

Stig Urheim, +5 more
- 01 May 2005 - 
TL;DR: A segmental myocardial work index as derived from strain Doppler echocardiography and invasive pressure can be estimated by SDE in combination with LV pressure and the orientation of the loop can be used to assess whether the segment is active or passive.
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Mechanism of prolonged electromechanical delay in late activated myocardium during left bundle branch block.

Kristoffer Russell, +9 more
- 07 Oct 2011 - 
TL;DR: It is concluded that, during LBBB, prolonged EMD in late-activated myocardium is caused by a higher dP/dt at the time of activation, resulting in aggravated mechanical relative to electrical dyssynchrony.