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Irmgard Th. Lippe
Researcher at Medical University of Graz
Publications - 67
Citations - 2571
Irmgard Th. Lippe is an academic researcher from Medical University of Graz. The author has contributed to research in topics: Receptor & Capsaicin. The author has an hindex of 28, co-authored 67 publications receiving 2488 citations. Previous affiliations of Irmgard Th. Lippe include University of Graz & University of the Witwatersrand.
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Klebsiella oxytoca as a causative organism of antibiotic-associated hemorrhagic colitis.
Christoph Högenauer,Cord Langner,Eckhard Beubler,Irmgard Th. Lippe,Rudolf Schicho,Gregor Gorkiewicz,Robert Krause,Nikolas Gerstgrasser,Guenter J. Krejs,Thomas A. Hinterleitner +9 more
TL;DR: The fulfillment of Koch's postulates for cytotoxin-producing K. oxytoca suggests that it is the causative organism in at least some cases of antibiotic-associated hemorrhagic colitis.
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Afferent nerve-mediated protection against deep mucosal damage in the rat stomach
Peter Holzer,M.A. Pabst,Irmgard Th. Lippe,Brigitta M. Peskar,Bernhard A. Peskar,Edward H. Livingston,Paul H. Guth +6 more
TL;DR: Data indicate that afferent nerve stimulation by intragastric capsaicin protects against deep mucosal damage in response to ethanol, an effect that seems related to an increase in mucosal blood flow but not to eicosanoid formation.
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The effect of omega conotoxin GVIA, a peptide modulator of the N-type voltage sensitive calcium channels, on motor responses produced by activation of efferent and sensory nerves in mammalian smooth muscle.
Carlo Alberto Maggi,Riccardo Patacchini,Paolo Santicioli,Irmgard Th. Lippe,Sandro Giuliani,Pierangelo Geppetti,Elena Del Bianco,Silvia Selleri,Alberto Meli +8 more
TL;DR: These findings indicate that CTX-sensitive calcium channels modulate transmitter release in autonomic nerve terminals of mammals, but noticeable species and organ related variations exist in sensitivity to this peptide, possibly reflecting the existence of a heterogenous population of voltage- sensitive calcium channels.
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Participation of endothelium-derived nitric oxide but not prostacyclin in the gastric mucosal hyperaemia due to acid back-diffusion.
Irmgard Th. Lippe,Peter Holzer +1 more
TL;DR: Findings indicate that endothelium‐derived NO plays an important mediator role in the gastric mucosal vasodilatation caused by back‐diffusion whilst vasodilsator prostanoids such as prostacyclin are not involved.
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Increased expression of TRPV1 receptor in dorsal root ganglia by acid insult of the rat gastric mucosa.
TL;DR: The TRPV1 receptor may be a key molecule in the transduction of acid‐induced nociception of the gastric mucosa and a mediator of visceral hypersensitivity.