J
Jingying Pan
Researcher at Nantong University
Publications - 6
Citations - 32
Jingying Pan is an academic researcher from Nantong University. The author has contributed to research in topics: Medicine & Spinal cord injury. The author has an hindex of 1, co-authored 1 publications receiving 6 citations.
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Journal ArticleDOI
Epitranscriptomic m6A regulation following spinal cord injury
Lingyan Xing,Yunyun Cai,Tuo Yang,Weiwei Yu,Mengdie Gao,Rui Chai,Sujun Ding,Jinhuan Wei,Jingying Pan,Gang Chen +9 more
TL;DR: M6A RNA methylation is dynamic and conserved following SCI and may contribute to spinal cord regeneration, and the expression of METTL3 is increased in both astrocytes and neural stem cells.
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Knockdown of polypyrimidine tract binding protein facilitates motor function recovery after spinal cord injury
Riyun Yang,Rui Chai,Jingying Pan,Jing-Yin Bao,Panhui Xia,Yan-Kai Wang,Ying Chen,Yi Li,Jian Wu,Gang Chen +9 more
TL;DR: Findings suggest that PTB knockdown may be a promising therapeutic strategy to promote motor function recovery during spinal cord repair.
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HAP1 inhibition contributes to neuropathic pain by suppressing Cav1.2 activity and attenuating inflammation.
Jingying Pan,Ya-Yu Zhao,Rui Sang,Riyun Yang,Jingyin Bao,Yongjiang Wu,Ying Fei,Jian Wu,Gang Chen +8 more
TL;DR: Wang et al. as mentioned in this paper showed that HAP1 level was greatly increased in the dorsal horn and dorsal root ganglia under acute and chronic pain conditions, which indicated that deficiency might attenuate pain responses.
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Application and prospects of somatic cell reprogramming technology for spinal cord injury treatment
TL;DR: Somatic cell reprogramming is a promising technology that has gradually become a feasible therapeutic approach for repairing the injured spinal cord. as discussed by the authors provides an overview of the transcription factors, genes, microRNAs (miRNAs), small molecules and combinations of these factors that can mediate somatic cell re-rogramming.
Journal Article
[Coxsackie virus B3 (CVB3) triggers the activation of NLRP3 in mouse macrophages by activating nicotinamide adenine dinucleotide kinase 2 (NADK2)].
TL;DR: In this article , the effects of Coxsackie virus B3 (CVB3) on the NLR family, pyrin domain containing protein 3 (NLPR3) of mouse macrophages and its mechanisms were examined.