J
Joan M. Broquetas
Researcher at Autonomous University of Barcelona
Publications - 54
Citations - 2054
Joan M. Broquetas is an academic researcher from Autonomous University of Barcelona. The author has contributed to research in topics: Respiratory muscle & Respiratory function. The author has an hindex of 20, co-authored 54 publications receiving 1947 citations. Previous affiliations of Joan M. Broquetas include Services Hospital & Pompeu Fabra University.
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Journal ArticleDOI
Inspiratory muscle training in patients with chronic obstructive pulmonary disease: structural adaptation and physiologic outcomes.
Alba Ramírez-Sarmiento,Mauricio Orozco-Levi,Rosa Güell,Esther Barreiro,Nuria Hernández,Susana Mota,Merce Sangenis,Joan M. Broquetas,Pere Casan,Joaquim Gea +9 more
TL;DR: It is demonstrated that inspiratory training induces a specific functional improvement of the inspiratory muscles and adaptive changes in the structure of external intercostal muscles.
Journal ArticleDOI
Injury of the human diaphragm associated with exertion and chronic obstructive pulmonary disease.
TL;DR: It is concluded that sarcomere disruption is common in the human diaphragm, is more evident in patients with COPD, and is higher after inspiratory loading, especially in the diaphragem of those with COPd.
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Subcellular adaptation of the human diaphragm in chronic obstructive pulmonary disease
Mauricio Orozco-Levi,Joaquim Gea,Josep Lloreta,Miquel Felez,J. Minguella,Sergi Serrano,Joan M. Broquetas +6 more
TL;DR: Impaired lung function occurring in patients with chronic obstructive pulmonary disease is associated with subcellular changes in their diaphragm, namely a shortening in the length of sarcomeres and an increase in the concentration of mitochondria, probably contributing to a better functional muscle behaviour.
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Metabolic characteristics of the deltoid muscle in patients with chronic obstructive pulmonary disease
TL;DR: Chronic obstructive patients show a preserved or even increased (severe disease) oxidative capacity in their deltoid muscle, which coexists with a greater capacity in the anaerobic part of the glycolysis.
Journal ArticleDOI
Oxidative stress, redox signaling pathways, and autophagy in cachectic muscles of male patients with advanced COPD and lung cancer.
Ester Puig-Vilanova,Diego A. Rodríguez,Josep Lloreta,Pilar Ausín,Sergio Pascual-Guardia,Joan M. Broquetas,Josep Roca,Joaquim Gea,Esther Barreiro,Esther Barreiro +9 more
TL;DR: Oxidative stress and redox-sensitive signaling pathways are likely to contribute to the etiology of muscle wasting and sarcomere disruption in patients with respiratory cachexia: LC and COPD.