J
John Bannigan
Researcher at University College Dublin
Publications - 61
Citations - 1710
John Bannigan is an academic researcher from University College Dublin. The author has contributed to research in topics: Embryo & Omphalocele. The author has an hindex of 21, co-authored 61 publications receiving 1623 citations. Previous affiliations of John Bannigan include National University of Ireland & Our Lady's Children's Hospital.
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Journal ArticleDOI
Cadmium : Toxic effects on the reproductive system and the embryo
Jennifer Thompson,John Bannigan +1 more
TL;DR: The mechanisms by which Cd can affect reproductive health are examined, and the use of micronutrients in prevention of these problems is considered.
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Teratogenic and developmental effects of lithium.
James J. Giles,John Bannigan +1 more
TL;DR: The results of various studies including cohort, prospective, retrospective and small number case reports indicate that lithium is a "weak" teratogen in humans, and experiments with other vertebrates have shown that lithium affects dorsoventral specification and inhibition of vasculogenesis.
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Stereology of the placenta in type 1 and type 2 diabetes
TL;DR: This study demonstrates an association between maternal diabetes and increased terminal villous volume and capillary volume and length is increased in the placentae of normally grown infants of T1DM diabetic mothers compared to non-diabetic controls, suggesting that factors other than glycaemia have a role in placental development in pre-gestational diabetes.
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Ethanol teratogenicity in mice: A light microscopic study
John Bannigan,Paul E. Burke +1 more
TL;DR: It is likely that the toxicity of ethanol is exerted primarily on some component of the cytoplasm and not on DNA synthesis, as many ethanol-treated embryos had open defects of the cranial neural tube.
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Effects of cadmium on cell death and cell proliferation in chick embryos.
TL;DR: It is hypothesized that the ventral body wall defect resulting from Cd treatment in chick embryos is the result of changes in the somites perhaps following interruption of a signalling pathway originating in ectoderm.