J
John Kingdom
Researcher at Lunenfeld-Tanenbaum Research Institute
Publications - 13
Citations - 553
John Kingdom is an academic researcher from Lunenfeld-Tanenbaum Research Institute. The author has contributed to research in topics: Preeclampsia & Pregnancy. The author has an hindex of 7, co-authored 13 publications receiving 384 citations. Previous affiliations of John Kingdom include University of Toronto & University Health Network.
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Journal ArticleDOI
Vascular Dysfunction in Women With a History of Preeclampsia and Intrauterine Growth Restriction Insights Into Future Vascular Risk
Yoav Yinon,John Kingdom,John Kingdom,Ayodele Odutayo,Rahim Moineddin,Sascha Drewlo,Vesta Lai,David Z.I. Cherney,Michelle Hladunewich +8 more
TL;DR: In this article, a study was conducted to assess vascular function in postpartum women and to determine whether differences emerged depending on the presentation of placental disease and whether preexisting endothelial dysfunction underlie both the predisposition to placental diseases and the later development of vascular disease.
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Screening for fetal growth restriction and placental insufficiency.
TL;DR: New approaches to effective screening for FGR are highlighted based on a comprehensive review of: etiology, diagnosis, antenatal surveillance and management, and the new maternal blood biomarker placenta growth factor.
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Low Molecular Weight Heparin Improves Endothelial Function in Pregnant Women at High Risk of Preeclampsia.
TL;DR: In this paper, the authors investigated the cardiovascular effects of low molecular weight heparin (LMWH) in pregnant women at high risk of severe preeclampsia, although the mechanisms of action are unknown.
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PPAR- γ Regulates Trophoblast Differentiation in the BeWo Cell Model
TL;DR: It is shown that blocking PPAR-γ activity induces cell proliferation at the expense of differentiation, while these remain unaltered following treatment with the agonist rosiglitazone.
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Should Maternal Hemodynamics Guide Antihypertensive Therapy in Preeclampsia
TL;DR: Preeclampsia is considered a more severe disease that progresses rapidly and is associated with increased risk of significant maternal and perinatal adverse outcomes, including placental abruption, preterm birth, and low birthweight.