J
John S. Bevan
Researcher at University of Wales
Publications - 5
Citations - 533
John S. Bevan is an academic researcher from University of Wales. The author has contributed to research in topics: Pituitary tumors & Pituitary neoplasm. The author has an hindex of 5, co-authored 5 publications receiving 515 citations.
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Journal ArticleDOI
Dopamine agonists and pituitary tumor shrinkage
TL;DR: Preliminary evidence suggests that dopamine agonists may restrain the growth of some functionless tumors; most of these tumors, however, can be satisfactorily debulked using transsphenoidal surgery, although the number of tumors studied is small.
Journal ArticleDOI
Low recurrence rate after partial hypophysectomyfor prolactinoma: the predictive value ofdynamic prolactin function tests
Jonathan Webster,M. D. Page,John S. Bevan,Stephen H. Richards,Anthony Douglas-Jones,Maurice F. Scanlon +5 more
TL;DR: To determine the factors influencing the outcome of transethmoidal partial hypophysectomy for suspected prolactinoma and the predictive value of pre and post‐operative dynamic PRL function tests.
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Preliminary characterization of growth factors secreted by human pituitary tumors.
TL;DR: The findings suggest that cells derived from human pituitary adenoma tissue synthesize and secrete several growth factors, each of which may have its own target cell specificities.
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Cholinergic blockade with pirenzepine improves carbohydrate tolerance and abolishes the GH response to meals in normal subjects.
TL;DR: The data suggest that the effect of pirenzepine on the glucose response to meals is at least partly independent of the inhibition of GH release, and is of relevance to the further investigation of cholinergic muscarinic antagonist in diabetes mellitus.
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Growth factors and pituitary tumors.
TL;DR: Adenoma formation may result from abnormal production of factors or their specific cellular receptors, loss of local inhibitory influences, activation of the intracellular secondary message pathways conveying the mitogenic signal to the nucleus, or deregulation of the nuclear processes controlling mitosis.