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Jonathan E. Efron

Researcher at Johns Hopkins University

Publications -  72
Citations -  2263

Jonathan E. Efron is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Colorectal cancer & Abdominal surgery. The author has an hindex of 21, co-authored 72 publications receiving 2029 citations. Previous affiliations of Jonathan E. Efron include Mayo Clinic & University of North Carolina at Chapel Hill.

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Readmission rates and cost following colorectal surgery.

TL;DR: Readmission after colorectal surgery occurs frequently and is associated with a cost of approximately $9000 per readmission, and clinical and systems-based prevention strategies are needed to reduce readmission.
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Patient readmission and mortality after colorectal surgery for colon cancer: impact of length of stay relative to other clinical factors.

TL;DR: Readmission rates after colectomies have increased during the past 2 decades and mean LOS after this operation has declined, and more research is needed to understand the balance and possible trade off between these hospital performance measures for all surgical procedures.
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Implementation of a Surgical Comprehensive Unit-Based Safety Program to Reduce Surgical Site Infections

TL;DR: A surgery-based CUSP intervention that might have markedly decreased SSI in a high-risk population is demonstrated and Formation of small groups of front-line providers to address patient harm using local wisdom and existing evidence can improve patient safety.
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Early experience with the bioabsorbable anal fistula plug.

TL;DR: The use of the Surgisis® anal plug was associated with a low rate of fistula healing and a high incidence of perianal sepsis, and the addition of a transanal advancement flap to the procedure may improve success rates.
Journal Article

Wound healing in nude mice: a study on the regulatory role of lymphocytes in fibroplasia.

TL;DR: The data suggest that T cells play a dual role in wound healing: an early stimulatory role on macrophages, endothelial cells, and fibroblasts, and a late counterregulatory role, which may be responsible for the orderly completion of wound repair.