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Juan Ybarra

Researcher at Hospital de Sant Pau

Publications -  49
Citations -  1623

Juan Ybarra is an academic researcher from Hospital de Sant Pau. The author has contributed to research in topics: Diabetes mellitus & Population. The author has an hindex of 21, co-authored 49 publications receiving 1509 citations. Previous affiliations of Juan Ybarra include Chartered Institute of Management Accountants & Autonomous University of Barcelona.

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Unchanged Hypovitaminosis D and Secondary Hyperparathyroidism in Morbid Obesity after Bariatric Surgery

TL;DR: It seems advisable to support vitamin D supplementation in the morbidly obese population and Hypovitaminosis D with secondary hyperparathyroidism due to low calcidiol bio-availability should be added to the crowded list of sequelae of morbid obesity.
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PTEN promoter methylation in sporadic thyroid carcinomas.

TL;DR: A high frequency of PTEN promoter hypermethylation is shown, especially in follicular tumors, suggesting its possible role in thyroid tumorigenesis.
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Stem cells isolated from adipose tissue of obese patients show changes in their transcriptomic profile that indicate loss in stemcellness and increased commitment to an adipocyte-like phenotype

TL;DR: The transcriptomic profile of the stem cells reservoir in obese subcutaneous WAT is highly modified with significant changes in genes regulating stemcellness, lineage commitment and inflammation.
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The subcutaneous adipose tissue reservoir of functionally active stem cells is reduced in obese patients

TL;DR: It is reported that subcutaneous WAT stromal vascular fraction from nonobese individuals had a significantly higher number of CD90+ cells than SVF from obese patients, and ADSCs in adipose tissue of obese patients have lower capacity for spontaneous or therapeutic repair.
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A new table for prevention of hypoglycaemia during physical activity in type 1 diabetic patients

TL;DR: It is shown that by replacing adequately the carbohydrates during the practice of physical exercise it is possible to prevent almost all hypoglycaemia episodes, independently of the insulin dosage adjustments.