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Jumina Jumina

Researcher at Gadjah Mada University

Publications -  92
Citations -  509

Jumina Jumina is an academic researcher from Gadjah Mada University. The author has contributed to research in topics: Adsorption & Chemistry. The author has an hindex of 10, co-authored 77 publications receiving 323 citations.

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Biological activity, quantitative structure-activity relationship analysis, and molecular docking of xanthone derivatives as anticancer drugs.

TL;DR: Xanthone formed binding interactions with some receptors involved in cancer pathology, including telomerase, tumor-promoting inflammation (COX-2), and cyclin-dependent kinase-2 (CDK2) inhibitor, and this equation model generated 15 proposed new xanthone compounds with better-predicted anticancer activities.
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Preparation of Monoacylglycerol Derivatives from Indonesian Edible Oil and Their Antimicrobial Assay against Staphylococcus aureus and Escherichia coli

TL;DR: The preliminary antibacterial assay shows that the 1-monolinolein did not give any antibacterial activity against Staphylococcus aureus and Escherichia coli, while 2-monoolein showed weak antib bacterial activity againstStaphylitis aureUS.
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In Vitro Antifungal Activity of (1)-N-2-Methoxybenzyl-1,10-phenanthrolinium Bromide against Candida albicans and Its Effects on Membrane Integrity.

TL;DR: In vitro antifungal activity of FEN and its effect on the membrane integrity of Candida albicans indicated that membrane disruption is the probable mode of action of this compound.
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Antibacterial and Antifungal Activity of Three Monosaccharide Monomyristate Derivatives.

TL;DR: Amongst the esters, galactosyl myristate yielded the highest antibacterial activity against Salmonella typhimurium, Staphylococcus aureus and Bacillus subtilis, while glucosyl monomyristate exhibited the highest antibiotics activity only against Escherichia coli.
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Additive In Vitro Antiplasmodial Effect of N-Alkyl andN-Benzyl-1,10-Phenanthroline Derivatives and Cysteine Protease Inhibitor E64

TL;DR: Results indicated that these new compounds had a mechanism of action by inhibiting Plasmodium proteases, which are potential new targets for antimalarial chemotherapy include parasite proteases.