J
Jun-Liang Chen
Researcher at Jiangnan University
Publications - 22
Citations - 403
Jun-Liang Chen is an academic researcher from Jiangnan University. The author has contributed to research in topics: Lung injury & Heme oxygenase. The author has an hindex of 9, co-authored 22 publications receiving 233 citations. Previous affiliations of Jun-Liang Chen include Nanjing Medical University.
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Journal ArticleDOI
Nrf2 protects against seawater drowning-induced acute lung injury via inhibiting ferroptosis.
Yu-bao Qiu,Bin-bin Wan,Gang Liu,Ya-Xian Wu,Dan Chen,Mu-dan Lu,Jun-Liang Chen,Renqiang Yu,Daozhen Chen,Qing-feng Pang +9 more
TL;DR: Results suggested that Nrf2 can inhibit ferroptosis and therefore alleviate ALI induced by seawater drowning, providing a novel therapeutic target for seaw water drowning-induced ALI.
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Exogenous Hydrogen Sulfide Postconditioning Protects Isolated Rat Hearts From Ischemia/Reperfusion Injury Through Sirt1/PGC-1α Signaling Pathway.
TL;DR: H2S confers protective effects against I/R injury through the activation of Sirt1/PGC1α, and treatment with EX-527 could partially prevent the above effects of NaHS postconditioning.
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Protective effects of caffeic acid phenethyl ester against acute radiation-induced hepatic injury in rats.
TL;DR: Pretreatment with CAPE offers protection against radiation-induced hepatic injury in rats, and TUNEL assay showed that CAPE pretreatment inhibited hepatocyte apoptosis.
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Caveolin-1 scaffolding domain peptides enhance anti-inflammatory effect of heme oxygenase-1 through interrupting its interact with caveolin-1
Ping Weng,Xiao-Tong Zhang,Qiong Sheng,Wen-Fang Tian,Jun-Liang Chen,Jia-Jia Yuan,Ji-Ru Zhang,Qing-feng Pang +7 more
TL;DR: Caveolin-1(Cav-1) scaffolding domain peptides have beneficial anti-inflammatory effects by restoring the HO-1 activity suppressed by Cav-1 on plasma membrane.
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Hyperoside suppresses hypoxia-induced A549 survival and proliferation through ferrous accumulation via AMPK/HO-1 axis.
TL;DR: Taken together, the results highlighted the beneficial role of Hy against hypoxia-induced A549 survival and proliferation through ferrous accumulation via AMPK/HO-1 axis.